Hippocampal infolding angle in patient and control groups Epilepsy Febrile convulsion Control (n=83) (n=49) (n=74) Right CP 76.65[+ or -]5.79 76.63[+ or -]5.96 79.86[+ or -]4.55 Left CP 75.14[+ or -]5.49 75.27[+ or -]5.94 77.54[+ or -]6.45 Right SCP 93.63[+ or -]4.63 93.92[+ or -]4.77 95.74[+ or -]5.41 Left SCP 91.86[+ or -]4.91 91.16[+ or -]6.5 93.56[+ or -]5.32 P Right CP 0.001 (a) 0.002 (b) 0.008 (c) Left CP 0.01 (a) 0.015 (b) 0.05 (c) Right SCP 0.017 (a) 0.018 (b) 0.09 (c) Left SCP 0.076 (a) 0.2 (b) 0.07 (c) CP, cerebral pedincule; SCP, superior cerebellar
The principal MRI findings of Joubert syndrome are deep interpeduncular fossa together with a narrowing of the isthmus, thickening of the superior cerebellar
peduncle, fourth ventricle deformity together with hypolplasia of the vermis, fastigumun rostral shift and sagittal vermian cleft originating from the incomplete union of the two halves of the vermis.
The superior cerebellar
artery (Table 2) supplies blood to the upper part of the cerebellum and the midbrain.[10,12] Brain attack of this vessel causes ipsilateral cerebellar ataxia, nausea, vomiting, slurred speech and contralateral loss of pain and thermal sensation.
(37.) Akhlaghi H, Corben L, Georgiou-Karistianis N, Bradshaw J, Storey E, Delatycki MB, Egan GF Superior cerebellar
peduncle atrophy in Friedreich's ataxia correlates with disease symptoms.
Saltzman type Termination site Pcom I Basilar artery between the SCA Hypoplasia and the AICA II Basilar artery above the origin of Patent the SCA IIIa Directly the SCA Patent Illb Directly the AICA Patent IIIc Directly the PICA Patent Pcom, posterior communicating artery; SCA, superior cerebellar
artery; AICA, anterior inferior cerebellar artery; and PICA, posterior inferior cerebellar artery.
The imminent pathology in the MVD responsible for the problem has been compression by an artery usually a Superior Cerebellar
Artery(SCA) or loop of Anterior Inferior Cerebellar artery(AICA), venous pathology implicated as a cause been reported in some series as high as 80-90% especially in the recurrent conditions and in the younger age group5-8.
There is severe hypogenesis of the cerebellar vermis, enlarged, horizontal superior cerebellar
peduncles, a small mid brain in the anteroposterior direction, and a "batwing" configuration of the mid-superior fourth ventricle (Figures 1 and 2).
A correlative proton MR Spectroscopy (1H MRS) exam was performed on the abnormal area in the right superior cerebellar
hemisphere using both short and long echo times (TE) of 144 and 35 ms, respectively (Figure 2).
The superficial temporal artery, occipital artery, and external carotid artery can be used to increase blood flow to superior cerebellar
artery, posterior cerebral artery, posterior inferior cerebellar artery, or anterior inferior cerebellar artery .
(1) This appearance is a result of absence or hypoplasia of the cerebellar vermis, lack of normal dorsal decussation and consequent enlargement of the superior cerebellar
peduncles which follow a more horizontal course as they extend perpendicularly to the brainstem between the midbrain and the cerebellum.
* like CN III, it passes between the posterior cerebral artery and superior cerebellar
This inhibitory effect is absent in cerebellar patients when lesions involve dentate nuclei or the superior cerebellar