In this current era of medical science, an increase in the use of interventional procedures such as central venous catheter insertion, implantable cardiac defibrillator, and cardiac pacemaker placement has caused a significant rise in the number of SVC syndrome of benign aetiology by predisposing to thrombosis.
The patient was hospitalized with a provisional diagnosis of SVC syndrome and was kept at head end elevated position with oxygen supplementation.
We present a case of antiphospholipid syndrome causing BCS with IVC and SVC thrombosis with progressive shortness of breath and hypoxemia due to SVC syndrome and SPVS.
First of all, it is an extremely rare benign cause of SVC syndrome and SPVS.
SVC syndrome reduces venous return to the heart and, like PE, may cause sinus tachycardia.
THROMBOLYSIS: Thrombolysis is indicated in young and healthy patients especially with primary UEDVT, symptomatic SVC syndrome, for preservation of a mandatory central venous catheter because they can have significant long-term morbidity if treated with conventional anticoagulation alone.
He was diagnosed with SVC syndrome secondary to a mediastinal mass, demonstrated best by computed tomography (CT) [Figures 1, 2, 3].
This case of SVC syndrome was due to an aggressive thyroid follicular carcinoma with extensive angioinvasion and venous spread.
The SVC syndrome
resolved with concurrent chemoradiotherapy before surgical intervention.
, constrictive pericarditis, dysphagia, odynophagia, and tracheoesophageal fistula might also result.
Nine months after initial presentation, he was readmitted for pneumonia, and it was noted at that time that he had recurrent facial and arm swelling with bilateral breast enlargement accompanied by an increase in the mediastinal mass and recurrent SVC syndrome.
The findings from hormonal analysis, improvement with anticoagulation, chemotherapy, and radiation, and return of breast enlargement with recurrence of the SVC syndrome point to venous obstruction as the cause.