S100A8, S100A9, S100A12
, and SAA were assessed by Western blot (WB) analysis.
 confirmed the relation between S100A12
concentration and the effectiveness of therapy.
Leach, "Fecal S100A12
in healthy infants and children," Disease Markers, vol.
induces the expression of endothelial adhesion molecules, promoting leukocyte recruitment (33), neutrophil oxidative burst, degranulation and secretion (34), and pulmonary mast cell activation (35).
Qureshi et al., "Elevated circulating S100A12
associates with vascular disease and worse clinical outcome in peritoneal dialysis patients," Peritoneal Dialysis International, vol.
Utter et al., "Inflammatory S100A9 and S100A12
proteins in Alzheimer's disease," Neurobiology of Aging, vol.
There was no relationship between fecal M2-PK and fecal S100A12
levels in the children with active CD.
: a noninvasive marker of inflammation in inflammatory bowel disease," Journal of Digestive Diseases, vol.
S100A8/A9 and S100A12
induce prothrombotic and proinflammatory responses in endothelial cells including induction of thrombospondin, chemokines, and adhesion molecules .
Several S100 proteins have so far been identified as DAMPs, including S100A7 , S100A8, S100A9, S100A12
[5, 7], and S100A15 .
A study in children undergoing cardiac surgery requiring CPB showed another leukocyte S100 protein, S100A12
, increased postoperatively and levels were correlated with respiratory parameters, ICU and hospital length of stay .
In recent years, the mediators S100A12
and RAGE have drawn attention as specific markers of ALI .