Raji cell

Ra·ji cell

a cell of a cultured line of lymphoblastoid cells derived from a Burkitt lymphoma; it possesses numerous receptors for certain complement components and is thus suitable for use in detection of immune complexes. It expresses certain complement receptors as well as Fc receptors for immunoglobulin G.
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These included assays in 2 tissues harboring confirmed sites of productive PV replication, tonsils and spinal cord, (13,14) and a human-derived tissue with confirmed CD155 absence, Raji cell xenografts (the CD155 gene is transcriptionally silenced in Epstein-Barr virus-infected lymphomas).
Therefore, in this study, an attempt was made to clone the cDNA coding for CD 19 molecule from Raji cell line for subsequent expression in mouse NIH-3T3 cell line and production of monoclonal antibodies against the extracellular part of molecule.
However, a simultaneous binaural bithermal test revealed an abnormal RVR left (figure 1), Moreover, the patient's response to a 5-hour glucose tolerance test was exaggerated, and his level of circulating immune complexes of the Raji cell type was markedly elevated.
sup][4] Raji cell line was used as a positive control.
However, its regulation of the human B lymphoma Raji cell cycle, cyclin and CDK inhibitor levels has not yet been investigated.
Monoclonal C3d antibody was used in the Cogent and Scimedx assays, whereas the Raji cell replacement assay used monoclonal C3bi and C3d.
Pompeia C, Boaventura MF, Cud R (2001) Antiapoptotic effect of dipyrone on HL-60, Jurkat and Raji cell lines submitted to UV irradiation, arachidonic acid and cycloheximide treatments.
The cell viability of all cells decreased with increasing concentration of wogonin, and the cell viability of MCL cells (Mino, JeKo-1, and REC-1 cells) decreased more notably compared with Jurkat (acute T lymphoblastic leukemia cell line) and Raji cells (Burkitt's lymphoma cell line) [Figure 1]a.
Anticancer activity: Showed a strong inhibition against tumor promoter 12-0 hexadecanoylphorbol-13 (HPA)-induced Epstein-Barr virus activation in Raji cells.
All eight substances also exhibited moderate inhibitory effects on Epstein-Barr virus early antigen (EBV-EA) in Raji cells as a primary screening test for tumor promoter inhibitors.
Jurkat and Raji cells were propagated in RPMI 1640 medium supplemented with 10% foetal bovine serum, 2 mM L-glutamine, 1 mM pyruvate, 10 mM HEPES, MEM Non-Essential Amino Acids 10[micro]/ml, 50[micro]g/ml penicillin and 50 [micro]g/ml streptomycin (all reagents from Life Technologies, Grand Island, NY, USA).

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