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Koltas et al suggested mean corpuscular volume (MCV) along with red cell distribution width (RDW) as a new parameter in the diagnosis of malaria.
Variables were compared using independent samples t-test for mean RDW, gender and presence of complications.
Receiver operating characteristic (ROC) curve analyses were conducted and areas under the ROC curves were calculated to evaluate the diagnostic accuracy of RDW and NLR in detecting lamina propria invasion.
In the third stage, the values of WBC, neutrophil count, platelet, MPV, RDW, total bilirubin, direct bilirubin, creatine, albumin, erythrocyte sedimentation rate, CRP, and neutrophil/lymphocyte ratio parameters were compared in order to predict the risk of bacteremia and third stage intensive care unit (ICU) among patients with pyelonephritis and urosepsis.
Age, gender, RDW value (reference range: 11.7%-14.6%), monocyte count (reference range: 0.3-0.9 x [10.sup.3]/[micro]L), hemoglobin value (reference range: 34.5%-46.3%), and the APACHE II score at 24 h after admission to the ICU were recorded in the admission of the patients to the ICU.
Elevated RDW or elevated RDW plus MPV increased the odds of atrial fibrillation, coronary artery disease, heart failure, and peripheral vascular disease anywhere from 2 to 8.3 times (P less than .001).
As shown in Table 2, the lymphocyte, platelet, and RDW values were significantly higher in group 1.
A majority of those excluded from the final sample were 1,216 individuals (10% of original data) with no recorded value for CRP, RDW, or both dependent variables.
This study aimed to evaluate serum RDW in patients with AS and to assess its relationships with APRs and AS disease activity index.
Of these patients, including 38 patients with RDW + NLR = 0, 37 patients with RDW + NLR = 1, and 28 patients with RDW + NLR = 2.
In this study, we analyzed the relationship between RDW and subclinical atherosclerosis measured by C-IMT and examined its potential role as a marker carotid atherosclerosis in Koreans with type 2 diabetes without CVD.
Charts were reviewed for lipid profiles (high density lipoproteins and total cholesterol), hemoglobin A1c (HbA1c), smoking history, diagnosis of hypertension (HTN), systolic blood pressure (SBP), antihypertensive usage, diagnosis of CVD, diagnosis of diabetes mellitus (DM), diagnosis of hyperlipidemia, RDW, CRP, hemoglobin, absolute lymphocyte count (ALC), albumin level, treatment with nonsteroidal anti-inflammatory drugs (NSAIDs), treatment with conventional synthetic disease-modifying antirheumatic drugs (csDMARDs), including methotrexate, sulfasalazine, and leflunomide, treatment with biologic monotherapy (including tumor necrosis factor inhibitors (TNF-I), interleukin-17 antagonists (IL-17A)), and combination biologic and csDMARD therapy.
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