long QT syndrome

(redirected from QT prolongation)

long QT syndrome

 
a combination of prolonged Q–T interval and torsades de pointes; it may be congenital or acquired, the latter usually being the result of drug administration.

long QT syndrome

an inherited cardiac disorder characterized by prolongation of the Q-T interval. The disorder is associated with ventricular tachycardia, cardiac arrhythmias, syncope, and sudden death. Syncopal episodes often occur during physical exercise in young, otherwise healthy persons. This syndrome may also be caused by a variety of drugs.

prolonged QT syndrome

, long QT syndrome, QT syndrome
A life-threatening syndrome marked by a prolonged Q-T interval with episodes of electrocardiographic torsades de pointes. This condition may be inherited or may be acquired as a result of drug administration. Inherited variants of the long QT syndrome include Romano-Ward syndrome and Lange-Nielsen syndrome. It is treated with beta-blocking drugs or an implanted cardioverter defibrillator (ICD).
References in periodicals archive ?
QT prolongation refers to the prolonged QT interval in electrocardiography (ECG) which can even lead to life-threatening events such as ventricular arrhythmias, torsades de pointes, etc.
Reference to alternative sources, such as the reference to an updated list of drugs to avoid in patients with QT prolongation, is also valuable.
Although QT prolongation is also related to innate variation in myocardial cation channel proteins (Arking et al.
5%) had dangerous QT prolongation - when heart muscle takes longer than normal to recharge between beats.
11, 19 Reduction of nitric oxide bioavailability and stimulation of protein kinase C were suggested as the mechanisms that cause QT prolongation in hyperglycemia.
In regard to cardiovascular risks, fluoroquinolones are associated with QT prolongation.
Moreover, increasing age was significantly associated with QT prolongation in females (p=0.
We hypothesized that QT prolongation should be common in patients with anemia if it can cause ALQTS directly, and aimed to test this hypothesis in patients with anemia caused by conditions other than sickle cell disease.
Reported AEs of hyperglycaemia, interstitial lung disease (ILD) and QT prolongation remained consistent with data previously presented: ILD and pneumonitis 3% (2% ?
QT prolongation and anticholinergic toxicity are further examples of additive PD drug-drug interactions.
Etiology of QT prolongation and T wave changes in chronic alcoholism.