Polycystic ovarian syndrome


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Polycystic ovarian syndrome (PCOS)

A condition in which the eggs are not released from the ovaries and instead form multiple cysts.
References in periodicals archive ?
Effects of metformin on inappropriate LH release in women with polycystic ovarian syndrome and insulin resistance.
Vitamin D level in unmarried females with Polycystic ovarian syndrome. JIIMC 2016; 11(2): 57-61.
Table 3 Acanthosis nigricans at different body areas in patients with polycystic ovarian syndrome (n=50).
Effect of yoga nidra on resting cardiovascular parameters in polycystic ovarian syndrome women.
Association between polycystic ovarian syndrome, overweight, and metabolic syndrome in adolescents.
Sattar, "Low grade chronic inflammation in women with polycystic ovarian syndrome," The Journal of Clinical Endocrinology and Metabolism, vol.
Sovio et al., "Large-scale analysis of the relationship between CYP11A Promoter variation, polycystic ovarian syndrome, and serum testosterone," The Journal of Clinical Endocrinology & Metabolism, vol.
Some doctors prescribe metformin (Glucophage) for treating prediabetes, particularly if there is a history of polycystic ovarian syndrome, but this should not be a substitute for a healthy lifestyle.
Mean age, mean gravidity, mean parity and duration of sexual life in patients with polycystic ovarian syndrome and the control group PCOS group Mean age (years) (mean + SD) 29.3[+ or -]5.2 (19-39) Mean gravidity (mean + SD) 1.7[+ or -]1.4 (1-5) Mean parity (mean + SD) 1.3[+ or -]1.1 (1-4) Duration of sexual lifea (mean + SD) 4.8[+ or -]3.6 years Control group p Mean age (years) (mean + SD) 27.9[+ or -]5.1 (21-35) 0.428 Mean gravidity (mean + SD) 2.5[+ or -]1.2 (0-4) 0.148 Mean parity (mean + SD) 1.8[+ or -]1.2 (0-4) 0.333 Duration of sexual lifea (mean + SD) 5.1[+ or -]3.1 years 0.778 (a) Duration of sexual life in years from initial coitus; p<0.05 PCOS: Polycystic ovarian syndrome, SD: Standard deviation Table 2.
Rodent models of polycystic ovaries have shown many characteristics similar to that in the human polycystic ovarian syndrome (PCOS) which includes hyper-androgenism, disrupted cyclicity, presence of follicular cysts/polycystic ovaries.5 Different androgens like Di-hydrotestosterone (DHT), estradiolvalerate, dehydro-epiandrosterone (DHEA), and testosterone were used to develop polycystic ovaries in rodents.5,6 In this study we used Testosterone propionate (TP) to develop a rodent model of the condition.
Polycystic Ovarian Syndrome (PCOS) is a condition that causes irregular menstrual periods with excessive facial and body hair growth, acne, and/or male pattern scalp hair thinning.
Polycystic ovarian syndrome (PCOS) is a very common endocrine system disorder affecting about 5-10% women of reproductive age.

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