In the early 1980s in Finland, serologic evidence associated SINV with rash and arthritis, known as Pogosta disease (19).
Causative agent of Pogosta disease isolated from blood and skin lesions.
We isolated SINV from five patients with acute Pogosta disease during an outbreak in fall 2002 in Finland.
The typical clinical picture of Pogosta disease includes arthritis, maculopapular rash, and sometimes low fever, fatigue, and muscle pain (4; Kurkela et al.
The aim of this study was to isolate the causative agent of Pogosta disease directly from human samples.
We collected samples from the health districts where the incidence of Pogosta disease had been highest during previous epidemic years, North Karelia and Kuopio.
Typical Pogosta disease symptoms began with polyarthritis, first affecting the fingers, wrists, and the left shoulder, then extending to the left elbow, and finally both knees and the right ankle.
We analyzed retrospectively the use of Physician Desk Reference Database searches to identify epidemics of tularemia, nephropathy, Pogosta disease, and Lyme disease and compared the searches with mandatory laboratory reports to the National Infectious Diseases Register in Finland during 1995.
5, caused by a Puumala virus (a hantavirus); Pogosta disease, ICD-10 A92.
The Lyme disease guideline was the third most frequently read, with 144 readings; Pogosta disease was fifth, with 91; epidemic nephropathy eighth, with 87; and tularemia nineteenth, with 68 readings.
A large epidemic of Pogosta disease took place in late summer and autumn 1995.
For tularemia (Figure, panel D) and Pogosta disease (Figure, panel C), the PDRD searches and the cases in NIDR rose from a low baseline level, peaked sharply, and then declined in parallel.