Pseudomonas aeruginosa

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Pseu·do·mo·nas ae·ru·gi·no·'sa

a bacterial species found in soil, water, and commonly in clinical specimens (wound infections, infected burn lesions, urinary tract infections); the causative agent of blue pus; occasionally pathogenic for plants; usually causes infections in humans in whom there is a defect in host defense mechanisms. It is the type species of the genus Pseudomonas.
Synonym(s): blue pus bacillus

Pseudomonas aeruginosa

Etymology: Gk, pseudes, false, monas, unity
a species of gram-negative, nonspore-forming, motile bacteria that may cause various human diseases ranging from purulent meningitis to nosocomial infected wounds. Also called Pseudomonas pyocyanea.

pseudomonas aeruginosa

A normal soil inhabitant and human saprobe/commensal which may contaminate various solutions and fluids in a hospital, causing opportunistic infection in immunocompromised patients.
 
Clinical findings
Infective endocarditis in IV drug users, respiratory tract infections, urinary tract infections, bacteraemia, meningitis, “malignant” external otitis.

Management
Aminoglycosides—e.g., gentamicin, amikacin, netilmicin, tobramycin, etc. 

Pathogenesis
Pseudonomas aeruginosa is both invasive and toxicogenic, and infects patients in a 3-step process:
1. Bacterial attachment and colonization—mediated by pili and antiphagocytic effects of the organism’s polysaccharide capsule;
2. Local invasion—mediated by elastase and bacterial alkaline protease; and
3. Dissemination—high-dose tobramycin delivered by aerosol is reportedly effective in patients with cystic fibrosis.

Pseudomonas aeruginosa

A normal soil inhabitant and human saprophyte that may contaminate various solutions in a hospital, causing opportunistic infection in weakened Pts Clinical Infective endocarditis in IVDAs, RTIs, UTIs, bacteremia, meningitis, 'malignant' external otitis Treatment Aminoglycosides–eg, gentamicin, amikacin, tobramycin, etc

Pseu·do·mo·nas ae·ru·gi·no·sa

(sū-dō-mōnăz ē-rū-ji-nōsă)
Bacterial species found in soil, water, and commonly in clinical specimens (wound infections, infected burn lesions, urinary tract infections); produces blue pus.
Enlarge picture
Psuedomonas Aeruginosa: infection of the distal foot

Pseudomonas aeruginosa

A species that produces a distinctive blue-green pigment, grows readily in water, and may cause life-threatening infections in humans, including nosocomial pneumonia, urinary tract infections, and sepsis. It may also cause folliculitis, malignant otitis externa, and skin infections in patients who have suffered burns. See: illustration
See also: Pseudomonas
Pseudomonas aeruginosa Gram-negative aerobic infection, with characteristic turquoise-green pus; pseudomonal infections respond to ticarcillin with clavulanic acid (Timentin)

Pseudomonas

a genus of gram-negative, strictly aerobic bacteria, some species of which are pathogenic for plants and vertebrates.

Pseudomonas aeruginosa
a common isolate from wounds, burns and urinary tract infections and from many other accumulations of pus in all species. Also commonly found in otitis externa, fleece rot in sheep, and some cases of bovine mastitis. Its presence may be indicated by a distinctive blue or green color of the pus or infected site.
Pseudomonas fluorescens
a common cause of food spoilage and a frequent isolate from wounds and other contaminated sites in animals.
Pseudomonas mallei
see burkholderia mallei.
Pseudomonas pseudomallei
see burkholderia pseudomallei.
Pseudomonas putida
causes septicemia in aquarium fish.
References in periodicals archive ?
Eleven types of 16S RMTases (ArmA, RmtA-RmtH, and NpmA) have so far been reported in several nosocomially transmitted pathogens, including P.
13-51 To our knowledge this was the first report of the detection of a VIM-2-producing P.
We believe that directing the human immune response to fight life-threatening infections such as those associated with Gram-negative P.
The aromadendrane-type sesquiterpenoid viridiflorol was the most potent biofilm formation inhibitor, producing 60% inhibition in P.
In order to more fully understand the role that environmental sampling methods play in the observed P.
The study compared nine strains against the standard P.
In a study, the team described how they solved the 3D structure of a receptor protein called PqsR used by P.
These microbes, dangerous in their own right, also can cause epithelial cell damage within the lung, which can increase attachment points for more microbes and eventually create ideal conditions for P.
Previous studies conducted at our medical center revealed the predominant prevalence of a potentially virulent P.
To explore the connection, the researchers infected mouse neutrophils with two clinical laboratory strains of P.
Although this study did not consider analysis under genomic level, it clearly proves the variation between different P.
As the phenotypic screening was negative for detecting MBLs producing isolates, the resistance to imipenem observed in four of them, is more likely due to a decrease in the expression of OprD, an outer membrane protein channel that acts as the passage for imipenem entry in P.