ornithine decarboxylase


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or·ni·thine de·car·box·yl·ase

an enzyme catalyzing the decarboxylation of l-ornithine to putrescine and CO2; first step in polyamine biosynthesis.
References in periodicals archive ?
Use of a modified ornithine decarboxylase promoter to achieve efficient c-MYC-or N-MYC-regulated protein expression," Cancer Research, vol.
Co-operation between follicular ornithine decarboxylase and v-Haras induces spontaneous papillomas and malignant conversion in transgenic skin.
In contrast, high concentrations of ornithine can inhibit the activity of ornithine decarboxylase due to the fast formation and accumulation of putrescine (DIRCKS et al.
k]B and ornithine decarboxylase, ARE induction and cell proliferation assays).
Kitani and Fujisawa (1988) indicated that the liver is characterized by a low ornithine decarboxylase (ODC) activity, which could explain the small variation in PAs levels.
Control of nucleic acid and protein synthesis in developing brain, kidney, and heart of the neonatal rat: effects of [alpha]-difluoromethylornithine, a specific, irreversible inhibitor of ornithine decarboxylase.
Analysis of ornithine decarboxylase messenger ribonucleic acid expression in colorectal carcinoma.
12129 13408 Serogroup O1 O22 Haemolysis on blood agar + + Indole + + Voges-Proskauer + + Lysine decarboxylase + + Arginine dihydrolase - - Ornithine decarboxylase + + Phage IV (lysis) - - Acid from: L-Arabinose - - Mannose + - Sucrose + + Growth in tryptone broth: 0% NaCl + + 3% NaCl + + Susceptibility to: O/129 (10 [micro]g disc) R R (150 [micro]g disc) R R Polymyxin B (50 U disc) R R Trimethoprim/sulphamethoxazole R R (1.
Yamakawa and Taira (1980) detected ornithine decarboxylase activity in B.
Arginine decarboxylase activity, but not ornithine decarboxylase, increased during water-deficit stress induced by various osmotica (Galston, 1989) or by withholding water (Flores and Galston, 1984b).
DFMO is an irreversible inhibitor of an important oncogene, ornithine decarboxylase (ODC).
Pretreatment with the mango constituent time- and dose-dependently inhibited multiple 12-0-tetradecanoyl-phorbol-13-acetate (TPA)-mediated increases in edema, hyperplasia, epidermal ornithine decarboxylase (ODC) activity, as well as protein expression of ODC, cyclooxygenase 2 (COX-2) and nitric oxide synthase.

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