OXT


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OXT

Abbreviation for oxytocin.

OXT

Abbreviation for oxytocin.
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The researchers also used functional magnetic resonance imaging--a technology that measures brain activity by detecting changes in oxygenated blood flow--to examine the regions of people's brains that were activated during various tasks, and they found that those with increased methylation of the OXT gene had reduced neural activity in brain regions associated with social-cognitive processing.
All of our tests indicate that the OXT gene plays an important role in social behavior and brain function," Haas said.
With respect to anxiety and arousal, in animal studies, OXT has been shown to potentiate the anxiolytic effect of diazepam through its action on the amygdale and inhibits the norepinepherine system's arousal of the sympathetic nervous system, while increasing parasympathetic tone.
War veterans with PTSD given one dose of OXT demonstrated decreased physiologic response to provoked combat memories.
Paraphrasing, one should be glad that pharmaceutical companies have neither found the means nor benefit in decreasing our OXT receptors, since then cooperation and care for kin would no longer be a moral value.
Estradiol enhances OXTR synthesis and post-receptor events mediated by OXT to ensure a PGF pulse frequency necessary for luteolysis.
Endometrial OXTR induced in the latter stages of the estrous cycle of ewes and cows, and a positive feedback loop between uterine PGF and OXT secretion by luteal cells induce pulsatile PGF secretion by increasing phospholipase C (PLC) activity leading to the production of inositol trisphosphate (IP3) and diacylglycerol (DAG).
Endometrial tissues from ovariectomized ewes receiving steroid replacement to permit OXT-induced increases in PGF secretion respond similarly to melittin, a stimulator of PLA2, and OXT (Lee and Silvia, 1994).
Further, aristolochic acid, an inhibitor of PLA2, inhibits the ability of melittin and OXT to increase PGF secretion.
1993) and OXT does not induce PLC activity in endometrial tissue from d 12, 14 and 16 of pregnancy (Silvia and Raw, 1993).
As noted previously, IFNT acts as a paracrine antiluteolytic hormone to abrogate development of the luteolytic mechanism dependent on actions of E2 and OXT by silencing expression of ESR1 and OXTR genes in uterine LE/sGE.
Inhibition of secretion of prostaglandins was cell-specific since secretion of PGF and PGE2 by stromal cells was not affected by either IFNT or OXT (Danet-Desnoyers et al.