NKCC1

NKCC1

This is an incorrect gene name for what is designated SLC12A2.
References in periodicals archive ?
Bumetanide, an inhibitor of NKCC1 (Na-K-2Cl cotransporter isoform 1), enhances propofol-induced loss of righting reflex but not its immobilizing actions in neonatal rats.
Blast-Induced Traumatic Brain Injury Triggered by Moderate Intensity Shock Wave Using a Modified Experimental Model of Injury in Mice
Focus on "COMMD1 interacts with the COOH terminus of NKCC1 in Calu-3 airway epithelial cells to modulate NKCC1 ubiquitination".
Association between rs6759298 and Ankylosing Spondylitis in Iranian Population
floridae genome database (http://genome.jgi-psf.org//Brafl1/Brafl1.home.html) for the homologues of vertebrate NKA and NKCC (using NKA alpha 1 and NKCC1 as queries) revealed the presence of a single NKA (GenBank accession number: XP_002610679.1) and NKCC (GenBank accession number: XP_002609755.1), respectively.
Activities of Amphioxus GH-Like Protein in Osmoregulation: Insight into Origin of Vertebrate GH Family
For example, glial cells alter the expression of KCC2 and NKCC1, thereby decreasing GABAergic inhibitory tone and increasing glutamate release [141].
Neuronal-Glial Interactions Maintain Chronic Neuropathic Pain after Spinal Cord Injury
Rafiqi et al., "Functional interactions of the SPAK/OSR1 kinases with their upstream activator WNK1 and downstream substrate NKCC1," The Biochemical Journal, vol.
Association between Cullin-3 Single-Nucleotide Polymorphism rs17479770 and Essential Hypertension in the Male Chinese Han Population
Our pilot data indicate that AVP inhibits the Na-K-2Cl cotransporter NKCC1 and activates the K-Cl cotransporter KCC2 in a manner consistent with reduction of excitability.My laboratory has an internationally leading role in work on neuronal pH and Cl- regulation and on functions of the immature brain.
PRESSBIRTH: Arginine vasopressin and ion transporters in the modulation of brain excitability during birth and birth asphyxia seizures
The main goal of this project is to understand the mechanisms that some ion transporters, like the NKCC1 protein, use to increase cell migration, and how different factors, like chemoattractive substances affect this process.
Dr. Alfredo Quinones: the brains behind brain cancer research
Anion ([Cl.sup.-)] secretion through the cystic fibrosis transmembrane conductance regulator (CFTR) or calcium-dependent chloride channels (CaCC) is enabled after building up a cytosolic anion potential, which results from the activation of basolateral transporters notably: the [Na.sup.+]/[K.sup.+]-ATPase maintaining the [K.sup.+] concentration gradient between the intracellular and the extracellular spaces; the [Ca.sup.2+]-dependent ([mathematical expression not reproducible]) and ATP- sensitive ([K.sub.ATP]) [K.sup.+] channels, responsible for the driving force allowing for uptake of Cl- via the basolateral [Na.sup.+]-[K.sup.+]-[Cl.sup.-] cotransporter type 1 (NKCC1).
Epithelial Electrolyte Transport Physiology and the Gasotransmitter Hydrogen Sulfide
The molecular mechanisms are well described in several species and include basolateral [Na.sup.+],[K.sup.+] -ATPase and [Na.sup.+],[K.sup.+],2[Cl.sup.-] cotransporter (NKCC1), apical cystic fibrosis transmembrane conductance regulator (CFTR), and cation leaky tight junctions between neighboring cells, which are structurally and functionally defined by various claudin isoforms.
Water transport and functional dynamics of aquaporins in osmoregulatory organs of fishes
Expression of GABA signaling molecules KCC2, NKCC1, and GAD1 in cortical development and schizophrenia.
Prenatal choline and the development of schizophrenia
Prolonged interferon-exposure decreases ion transport, NKCC1, and Na+-K+-ATPase expression in human intestinal xenografts in vivo.
Valoracion de parametros clinicos y lesiones en organos de cerdos durante el periodo posdestete
The basolateral [Na.sup.+]-[K.sup.+]-[2Cl.sup.-] co-transporter (NKCC1) is essential for secretion of fluid into the pulmonary airways.
Poster session: Nitschke Hall 9:00-11:00 AM
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