MIR23B

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Shi et al., "A lentivirus-mediated miR23b sponge diminishes the malignant phenotype of glioma cells in vitro and in vivo," Oncology Reports, vol.

MicroRNA in Glioblastoma: An Overview

Further studies showed that dormant phenotypes were induced by overexpression of miR23b in BM2 cells which suppressed MARCKS gene [51].

Mesenchymal Stem Cell-Derived Extracellular Vesicles: Roles in Tumor Growth, Progression, and Drug Resistance

For miRNA quantification, the following commercial miScript Primer Assays (Qiagen) were used: MIR23B, MIR28, MIR93, MIR122, MIR144, MIR200A, MIR212, MIR383, and MIR510.

mir23b correlated inverselywith CD3 and CD20 expressions (p = 0.00076; r = -0.523 and p = 0.0012; r = -0.507) and miR144 with CD3, CD20, and CD30 (p = 0.030; r = -0.352, p = 0.041; r = -0.333 and p = 0.0032; r = -0.47, resp.).

Redox Regulating Enzymes and Connected MicroRNA Regulators Have Prognostic Value in Classical Hodgkin Lymphomas

MiR23b controls TGF-beta1 induced airway smooth muscle cell proliferation via TGFbetaR2/p-Smad3 signals.

Inactivated Pseudomonas aeruginosa inhibits hypoxia-induced pulmonary hypertension by preventing TGF-[beta]1/Smad signaling

On the other hand, 4 different upregulated miRNAs (miR-17, miR-133b, miR-203, and miR23b) are more important for tDCs induction than aDCs and iDCs after 6 h of maturation.

MicroRNA profiling of activated and tolerogenic human dendritic cells

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