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pylori positivity was detected in 46.6% of the 1027 patients with inflammation, 74.4% of the 515 patients with neutrophil activity, 37.5% of the 48 patients with glandular atrophy, 58.3% of the 151 patients with intestinal metaplasia, and the 79.1% of 258 patients with lymphoid follicle (Table 2).
The lymphoid follicles revealed polyclonal positivity, showing the coexpression of CD3, CD5, and CD20 in different subpopulations of the same follicles (figure 3).
Interestingly, some investigators proposed lymphoid follicles to promote mucosal repair via the regulation of bone-marrow derived stem cell trafficking [28, 29].
(3) Histopathologically, the lesions are characterized by hyperplasia of lymphoid follicles, eosinophilic infiltrations and proliferation of fibrocollagenous tissue and post-capillary venules.
(1973) Pinocy tosis by epithelium associated with lymphoid follicles in the bursa of Fabricius, appendix, and Peyer's patches.
Orbital mass biopsy was performed and histopathological exam showed numerous lymphoid follicles with germinal centers, fibroid bundles and infiltration of IgG4+ plasma cells and eosinophils in lacrimal gland (Figure 2).
Multiple lymphoid follicles are found in the submucous membranes in the rats of 56-OBR group (Figure 2b).
With these findings open lung biopsy was performed and pathological examination revealed peribronchial lymphoid infiltration and lymphoid follicles which were compatible with FB (Figure 3a and b).
Hashimoto thyroiditis, first described by Hashimoto in 1912, is an autoimmune inflammation of the thyroid commonly affecting middle-aged women.1 Histologic features of HT include diffuse infiltration of lymphoid cells usually with formation of lymphoid follicles, varying degrees of fibrosis, oxyphilic change or squamous metaplasia in the epithelial cells.1 When the presence of focal lymphocytic infiltration is assumed to be an adequate criterion for diagnosis of HT, the incidence appears to be as high as 16-23% in elderly females.1
Antibodies to this protein stain the germinal center cells in lymphoid follicles, follicular cells, and interfollicular cells in follicular lymphoma, large B-cell lymphomas, and Burkitt's lymphoma, and the majority of the Reed-Sternberg cells in nodular lymphocyte-predominant Hodgkin's disease.
Hakaru Hashimoto, the researcher who first characterized HT, summarized the four histological characteristics of this disease as the formation of lymphoid follicles, marked changes in thyroid epithelial cells, extensive formation of new connective tissue, and diffuse round cell infiltration [4].