Farlex Partner Medical Dictionary © Farlex 2012
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The FV linked with the third ventricle via cerebral aqueduct (aqueduct of Sylvius), and caudally via the median aperture (foramen of Magendie) with vallecula cerebelli and cisterna magna with the spinal canal and laterally, via the lateral aperture (foramina of Luschka) with the cerebellopontine angles (Mavridis).
CSF passes through the ventricular system and exits from the fourth ventricle through the foramina of Luschka and Magendie, entering the contiguous subarachnoid space surrounding the brain and spinal cord.
It was first reported by Rokitansky, Luschka, and Wilks in the 19th century (1-4).
First described by Luschka in 1848 [1] and first reported in the English literature by Bailey in 1924 [2], it represents a variant (caudal extension) of cleft number 30 in Tessier's classification of craniofacial clefts [3].
In limited cases this complication may occur from injury of the so-called ducts of Luschka; this event can be identified either intraoperatively or postoperatively [2]: intraoperative recognition is rare because of the small calibre of the ducts of Luschka but requires immediate resolution to avoid further more invasive treatment.
En 1856, Hubert von Luschka realizo la primera descripcion de un cordoma en el clivus; sin embargo, Rudolf Virchow, en 1857, hizo su primera descripcion histologica y atribuyo su crecimiento a un origen cartilaginoso con degeneracion hidropica celular.
El anatomista Hubert von Luschka nacio en Konstanz al sur de Alemania, su libro de 3 volumenes de anatomia clinica se publico entre 1862 y 1867, y fue el primero en describir el estudio anatomico realizado en cadaveres humanos aparentemente sanos, mediante cortes y tecnicas de conservacion en los especimenes.
CP angle tumours in the fourth ventricle has a tendency to grow through the Foramen of Luschka into the CP angle.
Hemorrhage extended into the fourth ventricle tracking through the foramina of Luschka into the cistema magna and subarachnoid spaces.
The second mechanism involves large amounts of aneurysmal SAH regurgitating into the fourth ventricle via the foramina of Luschka. In this case, the source of the IVH was considered to be through the disrupted medial wall of the right temporal horn of the lateral ventricle.
Theories regarding the pathogenesis of the various BAs include the incomplete obliteration of the pharyngeal clefts, as suggested by Ascherson in 1832, (16) the precervical sinus theory suggested by His in 1886, (17) the thymopharyngeal theory put forth by Wenglowski in 1912, (18) and cystic degeneration of lymph nodes suggested by Luschka in 1848.