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Franz von, German anatomist, 1821-1908. See: Leydig cells, Leydig cell tumor, Sertoli-Leydig cell tumor.
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References in periodicals archive ?
Biallelic inactivation of the LHCGR causes Leydig cell hypoplasia (LCH, OMIM#238320) that leads to male disorders of sexual differentiation (DSD).
The submucosa consists of loose fibrous connective tissue containing many hemolymphatic vessels and Leydig cells.
It has been showed that the carbamate insecticide carbaryl has affected spermatogenic cells and caused leydig cells degeneration and altered serum testosterone and gonadotrophin levels (Shrivastava and Shrivastava, 1998).
Reserpina Control p <0,05 Peso al sacrificio 189,6 [+ o -] 7,9 177,1 [+ o -] 15,7 NO (gramos) Peso testiculos 1,13 [+ o -] 0,04 1,72 [+ o -] 0,63 NO (promedio en gramos) Porcentaje (%) 76,0 [+ o -] 2,0 65,1 [+ o -] 2,2 SI celulas de Leydig + a BDNF Porcentaje (%) 30,0 [+ o -] 1,9 55,4 [+ o -] 9,4 SI celulas de Leydig + a caspasa-3 Porcentaje (%) 81,4 [+ o -] 1,7 72,5 [+ o -] 1,7 SI celulas de Leydig + a PCNA Indice de 1,1 [+ o -] 0,07 1,3 [+ o -] 0,02 NO positividad en celulas de Leydig a RA Diametro de tubulos 520,4 [+ o -] 17,3 479,5 [+ o -] 12,7 NO seminiferos (micrometros) Medias y errores para las variables estudiadas en animales tratados con reserpina y controles.
Affected fetuses possess fully functioning Leydig and Sertoli cells but cannot convert testosterone to DHT and present with variable degrees of undermasculinized external genitalia, normal-to-high male concentration of testosterone, low or low-normal DHT, and increased testosterone/DHT ratio at baseline or after hCG stimulation test.
Leydig cells were obtained from 42-day-old male Swiss mice.
Notable abnormalities were the decline of Leydig cell population within the interstitial compartment and the sloughing of germ cells within the seminiferous tubules (Figure 2).
Tavassoli, "Hepatocytic differentiation in retiform Sertoli-Leydig cell tumors: distinguishing a heterologous element from Leydig cells," Human Pathology, vol.
Indeed, several studies carried out in humans and animals showed that most of the adipokines and their receptors are expressed in testis especially in seminiferous tubes and more specifically in Leydig and Sertoli cells and on spermatozoa themselves [16].
The authors of this study conclude that a hypogonadal state or Leydig cell failure can be induced with zinc deficiency altering testicular steroidogenesis.
NIS expression was localized in the lumen side of the seminiferous ducts and Leydig cells but not observed in Sertoli cells with immunohistochemical examination (6).