As being a small vessel disease, most of the ischemic events appears in the form of subcortical infarctions causing
lacunar syndrome clinically.
Dysarthria-clumsy hand syndrome is a well-known, but infrequent
lacunar syndrome, which generally occurs due to a focal lesion in the basis pontis that damages corticofugal fibers, as well as adjacent pontine neurons or their axons (1,2).
Lacunar syndrome (pure motor hemiparesis, pure sensory stroke, mixed sensorimotor, ataxic hemiparesis, dysarthria clumsy hand) without new cortical
Stroke etiology was classified according to the Banff classification as large-vessel disease (ipsilateral carotid stenosis [greater than or equal to] 70%, presumable local thrombosis of a large intracranial vessel, arterio-arterial embolism from aortic plaques/thrombi), small-vessel disease (clinical
lacunar syndrome and no lesion or subcortical lesion <1.5 cm on computed tomography or magnetic resonance imaging), cardioembolic (high-risk source of cardiac embolism), or of undetermined etiology.
Clinical Stroke Syndrome: TACs = Total Anterior Circulation Sysndrome PACs = Partial Anterior Circulation Syndrome POCs = Posterior Circulation Syndrome LACs =
Lacunar Syndrome BM = Bladder Management Table 5.
The study group comprised of 128 first-ever stroke patients, aged between 40 and 65 years, seen at either the emergency department or the neurology outpatient services, within a week after the onset of symptoms of the classic
lacunar syndromes (pure motor hemiparesis, pure sensory stroke, sensorimotor stroke, ataxic hemiparesis, and dysarthria-clumsy hand syndrome), and diagnosed with stroke classed as "small-vessel" by the Trial of ORG 10172 in Acute Stroke Treatment (TOAST) classification [13].
Furthermore, small cardioembolic emboli can cause
lacunar syndromes, acting as a confounding factor in the analysis of the relation between HHcy and stroke subtypes.
