A separate study, published in the journal Cell Reports, identified a crucial role for a protein called Kruppel-like factor 9 (Klf9) in the brain's response to stress.
Other studies suggest that Klf9 levels may affect males and females differently, providing additional areas for Klf9 research.
Specifically it has been found that proteins klf9
KLF6 and PPARg regulate the increased expression of this, it promotes the development of hepatic steatosis, condition in which triglycerides accumulate in the hepatocytes (Youssef & Badr).
demonstrated that in NIH3T3 cells, an excessive oxidative stress environment can lead to Nrf2-dependent activation of Kruppel-like factor 9 (Klf9), a ubiquitously expressed protein that regulates cell differentiation and promotes oxidative stress-induced cell death .
Bagati et al., "Nrf2 amplifies oxidative stress via induction of Klf9," Molecular Cell, vol.
miR-567 SPTBN1; DUSP1; BCHE; LPHN2 miR-141-3p H3F3B; TCEAL2; MYH10; LHFP; LPHN2; CCL2; KLF9
miR-454-3p DPYSL2; HOXA5; FKBP11; SRPX; EDN1; LDLR; CAV2; BMPR2; SLC2A1 SERPINE2; PLEKHC1; SPTBN1; H3F3B; miR-590-3p TMEM47; TIMP3; COL3A1; CXCL13; ETS2; CELSR3; LPL; SMAD6; BMPR2; LPHN2; SASH1.
Common variants at CDKAL1 and KLF9
are associated with body mass index in East Asian populations.
One particular molecule (dubbed "Kruppel-like-factor 9" or Klf9
) stood out in the samples, they say.
 investigated the expression of Kruppel-like factor 9 (KLF9
), a progesterone receptor interacting protein, in eutopic endometrium of women with and without endometriosis.