Neural stem cells induce bone-marrowderived mesenchymal stem cells to generate neural stemlike cells via juxtacrine
and paracrine interactions.
Download PDF Brochure@ http://www.marketsandmarkets.com/pdfdownload.asp?id=80206120 Based on signaling type, the market is segmented into paracrine, endocrine, autocrine, juxtacrine
, and other signaling types (intracrine and neuronal signaling).
Mazzanti et al., "Mesenchymal stromal cells affect cardiomyocyte growth through juxtacrine
Notch-1/Jagged-1 signalling and paracrine mechanisms: clues for cardiac regeneration," Journal of Molecular and Cellular Cardiology, vol.
It should be kept in mind, however, that adipokines are part of large neuroendocrine-immune-metabolic networks, and they exert their effects through complex endocrine, paracrine, autocrine, or juxtacrine
crosstalk mechanisms, which should be further investigated with system medicine approaches.
Delarosa et al., "Juxtacrine
activity of estrogen receptor a in uterine stromal cells is necessary for estrogen-induced epithelial cell proliferation," Scientific Reports, vol.
Moreover, a SCLC cell xenograft model using nude mice demonstrated that the HH pathway was activated in SHH-producing SCLC cells but not in surrounding non-SHH-producing cancer cells, suggesting that HH pathway activation was an autocrine and/or juxtacrine
loop in SCLC.
Cytokines are small, nonstructural proteins with low molecular weights having autocrine, paracrine, and juxtacrine
effects and very complex activities.
and paracrine interactions of rat marrow-derived mesenchymal stem cells, muscle-derived satellite cells, and neonatal cardiomyocytes with endothelial cells in angiogenesis dynamics.
The interleukin-15/interleukin-15 receptor system as a model for juxtacrine
and reverse signaling.
They may communicate directly through juxtacrine
signaling, over short distances through paracrine signaling and over large distances through endocrine signaling.
Mesenchymal stromal cells affect cardiomyocyte growth through juxtacrine
Notch-1/Jagged-1 signaling and paracrine mechanisms: Clues for cardiac regeneration.
In sporadic cancer, the specific response to TGF-[beta] during tumor progression will depend on the stage of carcinogenesis and the responsiveness of tumor cells, and can be attributed to both independent and interrelated factors including changes in: (1) TGF-[beta] expression; (2) receptor expression; (3) availability of downstream signaling components; (4) evasion of the immune response; (5) stimulation of inflammation; (6) presence of local and systemic factors (autocrine, endocrine, paracrine, juxtacrine
, or matricrine interactions); and (7) the recruitment of cell types that lead to advanced tumor growth or promote angiogenesis .