Irvine-Gass syndrome

Ir·vine-Gass syn·drome

(ĭr'vīn gahs),
macular edema, aphakia, and vitreous humor adherent to incision for cataract extraction.

Ir·vine-Gass syn·drome

(ĭr'vīn gahs sin'drōm)
Macular edema, associated with cataract surgery, and vitreous humor adherent to incision after cataract extraction.

Gass,

J. Donald M., U.S. ophthalmologist, 1928–.
Gass cannula
Gass cataract-aspirating cannula
Gass corneoscleral punch
Gass muscle hook
Gass scleral marker
Gass scleral punch
Irvine-Gass syndrome - see under Irvine

Irvine,

A. Ray, Jr., U.S. ophthalmologist, 1917–.
Irvine corneal scissors
Irvine operation
Irvine probe-pointed scissors
Irvine-Gass syndrome - macular edema, aphakia, and vitreous humor adherent to incision for cataract extraction.

oedema, cystoid macular (CMO) 

Oedema and cyst formation of the macular area of the retina. It may occur as a result of, or be associated with, systemic vascular disease, retinal vein occlusion, diabetic retinopathy, uveitis, hypertensive retinopathy, retinitis pigmentosa and following some ocular surgery such as vitreoretinal, photocoagulation, glaucoma procedures and especially cataract surgery. When cystoid macular oedema follows cataract surgery it is called the Irvine-Gass syndrome and it is sometimes accompanied by intraoperative vitreous loss or vitreous adhesion to the iris or to the corneoscleral wound. Visual acuity is affected initially but recovers in the majority of cases. In some cases antiinflammatory therapy may help in restoring visual acuity and in other cases the vitreous adhesion may be disrupted with a Nd-Yag laser.
References in periodicals archive ?
Patients with a previous history of uveitis, those who had uveitis before ocular procedure, and those with other possible causes of uveitis (including endophthalmitis [2] and Irvine-Gass syndrome [11] with isolated macular edema on OCT and/or on fluorescein angiogram without any other inflammatory signs such as vitritis and vasculitis) were not included.
FA confirmed also the absence of late staining of the optic disc and the absence of vein abnormalities excluding an Irvine-Gass syndrome or a branch retinal vein occlusion (Figures 1(c), 1(d), 1(e), and 1(f)).
Since there was no medical history of diabetes, arterial hypertension, or X-ray radiation and considering the absence of evidence of Irvine-Gass syndrome or retinal vein occlusion on FA examination we conclude the diagnosis of bilateral type 1 IMT.
Indeed type 1 IMT must be differentiated from secondary telangiectasia caused by other retinal vascular diseases especially retinal venous occlusions, diabetic retinopathy, radiation retinopathy, Irvine-Gass syndrome, or hypertensive retinopathy.
Contributed by ophthalmologists from Europe, the US, and Israel, the 19 chapters address the surgical and nonsurgical management of vitreoretinal disorders, including diabetic retinopathy; diabetic macular edema, with discussion of the use of anti-vascular endothelial growth factor drugs and diagnosis; proliferative diabetic retinopathy; the complications and management of diabetic vitrectomy; retinal venous occlusions; rhegmatogenous retinal detachment, including vitrectomy, scleral buckling materials, and pneumatic retinopexy; the use of prophylaxis for retinal detachment; retinal detachment due to giant tears or dialysis; macular hole surgery; vitrectomy for epiretinal membranes; Irvine-Gass syndrome; and endophthalmitis.
Serous macular detachment has only recently been recognized to occur in a significant number of eyes with macular pathology including diabetic retinopathy, retinal vein occlusion, Behcet disease, Irvine-Gass syndrome and pars planitis.
Key Words: Serous macular detachment, optical coherence tomography, cystoid macular edema, diabetic retinopathy, retinal vein occlusion, Behcet disease, Irvine-Gass syndrome, pars planitis
Parikakis, "Intravitreal ranibizumab for the treatment of Irvine-Gass syndrome," Ocular Immunology and Inflammation, vol.
Odrobina, "Treatment of cystoid macular edema with bevacizumab in course of Irvine-Gass syndrome," Klinika Oczna, vol.
PCMO was first described by Irvine in 1953 and demonstrated angiographically by Gass and Norton in 1966 leading to the term Irvine-Gass syndrome. Incidence peaks at four-to-six weeks post-operatively, but can occur any time between four and 16 weeks after surgery.