PET scan showed multiple hypermetabolic
splenic lesions and no pathologic uptake in any other organ or lymph nodes.
Secondary penile metastases demonstrate similar metabolic activity to the primary neoplasm, as was seen in our case, where the penile metastases, primary lung lesion, adrenal lesion, and right subscapularis lesion all demonstrated similar hypermetabolic
The tumor and its extradural extension showed a major hypermetabolic
activity on positron emission tomography (PET).
Nevertheless, the right superior pulmonary vein appeared occluded by a thrombus and in close contact with a hypermetabolic
(SUV max 9.5) hilar mass with a 5 cm diameter (Figure 2).
Postoperative PET-CT showed significant residual disease and two hypermetabolic
lesions at left pleura and retrocrural tissue.
Two F-18 fludeoxyglucose (FDG) positron emission tomography images taken at four months intervals showed intense hypermetabolic
lymph node involvement and an increase in FDG metabolism in the left bowel localization of the abdomen, in the left external iliac area, in the left lumbar, and in the left paravertebral area (Figure 1).
A large markedly hypermetabolic
central hepatic mass (14.0 x 8.5 x 8.5 cm) with likely central necrosis was identified, consistent with primary malignancy.
This pattern was repeated when the GMN t-maps revealed large domains of hypometabolic and hypermetabolic
cortices, with vision areas showing the strongest relative hypermetabolism (Figure 6(b)).
The PET-CT revealed a hypermetabolic
mass in the gastric anastomosis site along with hypermetabolic
activity in the superior mesenteric vein (SMV) suspected with tumor thrombus (Figure 1).
The diagnosis of Graves' disease required high FT4 and/or FT3, low TSH, clinical signs of hypermetabolic
state diffuse activity increase in thyroid scintigraphy, diffuse thyroid enlargement in the ultrasound and presence of ophthalmopathy.
We completed positron emission and computed tomography scans by using intravenous F-18 fluoro-2-deoxyglucose that detected hypermetabolic
foci in the right axilla, subpectoral, subcarinal, and para hilar regions.
In addition, poor healing from suboptimal tissue oxygenation in patients who are often in pain and hypermetabolic
(with increased oxygen extraction) from the surgical stress response, may increase the graft failure rate.