HIVAN


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Related to HIVAN: HAART, azotemia, Iban

HIVAN

An abbreviation for HIV-associated nephropathy (kidney disease resulting from infection with the human immunodeficiency virus.
References in periodicals archive ?
Based on currently available literature, the youngest age of presentation of a child with HIVAN was 4 months in a series published from our centre in Durban, KwaZulu-Natal, South Africa (SA).
HIV-associated nephropathy (HIVAN) Membranoproliferative glomerulonephritis Mesangial glomerulonephritis with immune deposits Minimal change disease Lupus-like glomerulonephritis Crystal-induced nephropathy Fanconi syndrome Allergic interstitial nephritis Thrombotic thrombocytopenic purpura/Hemolytic uremic syndrome Paraproteinemia Lymphoma Table 2.
(33) Studies from African countries regarding the susceptibility of Africans to HIVAN are scanty.
Since discussing each unique HIV-connected renal disease would require a great deal of time and depth, the focus of this article is the very unique and specific renal disease entity of HIVAN.
For patients diagnosed with HIVAN, highly active anti-retroviral therapy (HAART) is the single most important intervention.
Two (1.76%) patients with HIVAN were diagnosed in our study, one presented with AKI and the other with AKI on CKD and both had proteinuria.
HIVAN, the classic kidney disease of HIV infection and the most common cause of CKD in HIV infected individuals, is mostly seen in African American descent and is consistent with a strong genetic predisposition.
Treating patients with HIVAN using ART, corticosteroids, cyclosporin and/or angiotensin inhibitors may reduce proteinuria and preserve kidney function as reported by Elewa et al.
The most common complication is HIVAN, a disease characterized by massive nephrotic proteinuria (often more than 10 grams/day), absence of edema, and large echogenic kidneys seen on ultrasound.
Statistics in the USA estimate the incidence of HIVAN as 3.5-12%.
Early in the HIV epidemic, kidney disease emerged as HIV-associated nephropathy (HIVAN), first reported in African Americans and Haitian immigrants with AIDS in 1984 by a group at the State University of New York in Brooklyn.
Intriguingly, the podocytes from patients with collapsing variant of FSGS and HIV associated nephropathy (HIVAN) return to express the proliferation marker Ki-67 [41].