H reflex

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H re·flex

a monosynaptic reflex consistently obtained in normal adults only by electrically stimulating the tibial nerve, generally in the popliteal fossa, while recording from the gastrocnemius-soleus muscle group; similar to the clinically elicited Achilles reflex, except that the neuromuscular spindles are bypassed. Widely used in the EMG laboratory to diagnose S1 radiculopathies and polyneuropathies.

H reflex

[after Johann Hoffmann, who described it in 1918] See: reflex

H reflex

In electrodiagnostic studies of spinal reflexes, the time required for a stimulus applied to a sensory nerve to travel to the spinal cord and return down the motor nerve.
See: F response
See also: reflex
References in periodicals archive ?
Alterations of H-reflex amplitude are normally ascribed to modifications in alpha motoneuron excitability and/or changes in presynaptic inhibition (PSI) of primary muscle spindle afferents (Zehr, 2002).
In patients with CP, the neural modulation of soleus H-reflex suppression was proposed as the mechanism accounting for the improvement in functional gait pattern after treadmill training therapy [19].
In addition, SS may induce reduction of the H-reflex amplitude, decreasing spinal reflex excitability and reducing stretch reflex activation in MTU (Avela et al., 199).
- Driving Speed Measurement Program Package (NCV, MCV, F-wave, H-Reflex, Blink Reflex, Repetitive Stimulation),
Another hypothesis that supports our results concerns the Hoffmann-reflex (H-reflex).
The H-reflex as a tool in neurophysiology: its limitations and uses in understanding nervous system function.
In two of the 3 successfully conditioned subjects, the conditioned MEP increase consisted of within-session task-dependent adaptation and cumulative long-term change, similar to previous studies of H-reflex operant conditioning [34, 37, 38].
The present study has been undertaken to measure the NCV in the proximal segment of the spinal monosynaptic reflex (H-reflex).
Depression of the Hoffman reflex (H-reflex) is used to examine alpha motor neuronal presynaptic influences during exercise, vibration, fatigue, and recovery in response to either a short term or long term intervention [1-4].
Latency of the soleus H-reflex was normal, and reciprocal suppression by dorsal flexion of the foot remained.