Graves' disease


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Graves' disease

(grāvz)
n.
A condition usually caused by excessive production of thyroid hormone and characterized by an enlarged thyroid gland, protrusion of the eyeballs, a rapid heartbeat, and nervous excitability. Also called exophthalmic goiter.

Graves' disease

Basedow disease Endocrinology The most common cause of hyperthyroidism, due to excess thyroid stimulation by autoantibodies with diffuse toxic goiter Clinical Hyperthyroidism, exophthalmos, dermatopathy–painless, reddish lumpy rash on anterior leg, familial tendency, tachycardia, ↑ metabolic rate Risk factors Stress, smoking, RT to neck, medications–eg IL-2 and IFN-alpha, and viral infection Diagnosis Thyroid scan–diffuse ↑ uptake, ↑ TSI–Thyroid Stimulating Ig level. See Exophthalmic goiter, Primary hyperthyroidism.

Graves' disease

The most common form of hyperthyroidism, characterized by bulging eyes, rapid heart rate, and other symptoms.

Patient discussion about Graves' disease

Q. Is there a recommended natural cure for graves disease? I've been suffering from graves for a year now and really feel tired of it. I'm thinking of trying some natural medications cause the regular ones are dissapointing so far. Any experience with that??

A. I am not familiar with any natural supplements you can take for graves disease, furthermore, it is an illness where you should be on close monitoring of an endocrinologist, and have them tell you which medications you are allowed to take and what could make the situation worse. If regular treatment doesn't seem to help you much you should go for another check up to see how your thyroid is doing.

Q. Is there a relation between certain types of allergies and graves? I suffer from graves and have been suffering from allergies since I was real young. Are these two related somehow?? anyone else suffers from both?

A. Not that I know about. However, allergy is quite common, and Graves' disease is also not a rare disease, so it's not impossible for the two to appear in the same person.

Take care,

More discussions about Graves' disease
References in periodicals archive ?
Rapid initiation of treatment is important when managing patients with Graves' disease, because untreated patients have a higher risk of psychiatric illness, cardiac disease, arrhythmia, and sudden cardiac death.
(8.) Committee on Pharmaceutical Affairs, Japanese Society for Pediatric Endocrinology, and the Pediatric Thyroid Disease Committee, Japan Thyroid Association (Taskforce for the Revision of the Guidelines for the Treatment of Childhood-Onset Graves' Disease), Minamitani K, Sato H, Ohye H, Harada S, Arisaka O.
Vitamin D and Graves' Disease: A Meta-Analysis Update.Nutrients 2015; 7: 3813-27.
Subjects who were newly diagnosed with Graves' disease showed improvement in the diastolic function, RV systolic function and other structural parameters with 6 months thyrostatic therapy.
Newly-diagnosed Patients with Graves' disease, who consecutively presented in the Endocrinology clinic and were under the age of fifty years, without history of cardiovascular, collagenous, pulmonary diseases were enrolled.
Table-3: Association of CD28 at C/T-I3 + 17 and that of CD40 at C/T-1 with Graves' disease. P-value indicates he results of Chi-square test calculated to demonstrate the correlation between three genotypes of each single nucleotide polymorphism with the Graves' disease.
Wall, "A 63 kDa skeletal muscle protein associated with eye muscle inflammation in Graves' disease is identified as the calcium binding protein calsequestrin," Autoimmunity, vol.
Rundle, "Management of exophthalmos and related ocular changes in Graves' disease," Metabolism, vol.
Diagnosis of Graves' disease (GD) was based on clinical and biochemical features of hyperthyroidism, [technetium.sup.99] scintiscan, and histological features.
Therefore, the greater BID (apoptotic) expression in the thyrocytes and lower expression of MCL-1 (antiapoptotic) in lymphocytes may lead to the routine use of these drugs in therapy for Graves' disease, changing its indication.
All subjects were previously examined by an endocrinologist, and the laboratory diagnosis of Graves' disease was based on the finding of undetectable serum TSH and high blood thyroid hormone associated with the presence of circulating antibodies (TRAb).
Graves' disease is an autoimmune disease, affecting the thyroid, orbital and periorbital tissue functionality (1).