At the electrophysiological study
a VT of similar morphology was induced in the anterior septal site of RVOT.
They are generally benign; in rare situations, like in the presence of structural heart disease, they can have catastrophic consequences because of rapid heart rates.1 SVTs, if recurrent, can result in significant impairment in the quality of life.2 An electrophysiological study
(EPS) is recommended for patients with symptomatic, paroxysmal SVT.3 Often, long-term medical therapy for SVTs is ineffective because of recurrent episodes despite the use of medications.4 Radiofrequency catheter ablation (RFCA) has become the treatment of choice due to its high primary success rates and low complication rates.5-7 The field of clinical cardiac electrophysiology has evolved dramatically over the last 30 years, beginning with the first description of the His bundle (H) recording in 1969.8
Out of 62 patients, 18 (29%) patients had CTS in right hand, 12 (19%) had left CTS and bilateral CTS in 14 (23%) and normal electrophysiological study
were present in 18 (29%) out of 62 patients.
It was demonstrated by an electrophysiological study
that iCEB is equal to the cardiac wavelength l (10).
Patients without symptomatic BrS but have significant risk factors, such as positive family history, usually undergo further risk stratification with an invasive electrophysiological study
In most cases, the electrophysiological study
induces ventricular rhythm disorders.
A new level of resolution in the electrophysiological study
requires the analysis of the effect of filtering elements on the bioelectric activity of the signal recorded with nanoelectrodes without filters.
A baseline electrophysiological study
was performed prior to the wound closure.
The electrophysiological study
includes dermatomal somatosensory evoked potentials, electromyography, F-wave latencies, H-reflexes, and motor and sensory nerve conduction determinations^].
A-16-year old boy with a history of surgical palliation of d-transposition of the great arteries, a normal systolic ejection fraction, and symptomatic drug refractory atrial flutter was referred for an electrophysiological study
and ablation procedure.
Flank bulge has never been reported following PCNL; however, flank bulge is a known potential complication of flank incisions for various retroperitoneal surgical procedures and has been reported in the urological,[sup.12] vascular[sup.13] and neurosurgical literature.[sup.14,15] Flank bulge due to laxity of the anterolateral abdominal musculature may be caused by damage to intercostal nerves.[sup.14] In a cadaveric and electrophysiological study
, Fahim and colleagues showed that the most significant intercostal nerve contributions to the anterolateral wall came from the T11 and T12 nerves.[sup.14] They concluded that postoperative flank bulge was likely due to denervation of the abdominal musculature from injury to the T11 and T12 intercostal nerves.