EC cell


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EC cell

1. An embryonal carcinoma cell, which is a cultured cell line.
2. An enterochromaffin cell that secretes substance P and is found in the stomach and small intestine.
See also: cell
References in periodicals archive ?
In conclusion, our study demonstrated that propofol inhibited cell proliferation, migration, and invasion but promoted apoptosis by regulation of Sox4 in EC cells. Our results might provide a new insight for the treatment of EC.
For the immunofluorescence assay, the cocultured cells were observed through a fluorescence microscope to investigate the phagocytosis of EC cells by macrophages.
Caption: Figure 3: Overexpression of HMGA2 relieved the effect of miR-195 on proliferation and apoptosis in EC cell lines.
Gene Expression Analysis of Embryonic Lineage Commitment in the Course of RA-Induced Differentiation of ES and EC Cells. To investigate early embryonic lineages during RA-induced differentiation of ES R1 and EC F9 cells, gene expression of pluripotency (Oct4 and Nanog) and lineage markers (Mvh, Gata4, Pax6, Afp, and Bry) was studied (Figures 2 and 3).
In addition, the past reports indicated that EC cell hyperplasia is considered to have close correlation with [CD4.sup.+] T lymphocytes, especially the Th1/Th2 balance [14, 15, 44].
Serotonin (5-HT) is such an important neurotransmitter of gastrointestinal system, located mainly in the EC cells of gastric mucosa, which helps in mucus secreting process (Guha and Ghosh 1995; Hernandez et al.
Dating from the earliest studies, scientists called EC cells stem cells.
We presume that the EC cells take in just enough FFAs to promote the release of fatty glyceride.
In the study by Rodel et al., the adhesion of PBMCs to activated EA.hy926 EC cells was significantly reduced 24 hours after IR with 0.5 Gy [16].
The OCT4A isoform is known as a prominent factor that sustains self-renewal and pluripotency in ES and EC cells. Compared to OCT4A, OCT4B isoforms such as OCT4B-164, OCT4B-190 and OCT4B-265 cannot sustain sternness properties in mentioned cells and respond to the cell stresses.
During differentiation, the ES cells lost pluripotency markers, such as Oct4 and Nanog, more readily than EC cells and then gained some, but not all, differentiation markers with faster kinetics.