When the presence of DPN
was evaluated, [Q.sub.ave] in the DPN
presence was found to be 34% lower (p<0.05).
was diagnosed using previously published criteria based on presence of neuropathic symptoms, decreased/absent ankle reflexes, abnormal sensory and motor findings, and abnormal nerve conduction studies (1).
Versus those without DPN
, those with the condition were more likely to be older (mean, 52 vs.
A preliminary analysis found that just 10% of the patients had signs of DPN
, according to their self-reports.
The prevalence of DPN
was 19.7% and increased with age and duration of diabetes.2 In a study from 2012 in the outpatient department of a tertiary care hospital the prevalence of DPN
was carried out using the NLP 2000 system (NanoInk, Inc., USA).
32 patients with DPN
and 12 age-matched control subjects underwent nerve conduction studies and assessment of corneal nerve morphometry at baseline and after approximately one year.
Specifically, E2 and ER[alpha] agonist PPT similarly reduced cell number by 75.90 [+ or -] 2.09% and 76.36 [+ or -] 1.05%, respectively; while ER[beta] agonist DPN
further reduced cell number to 7.03 [+ or -] 0.82% compared to the control group.
The aim of this study was to analyze postural stability, walking speed and fear of falling to determine predictors of falls in a group of patients with DPN
, as well as the impact of DPN
characteristics as confounding factors.
Given that, DPN
affects about 60 to 70 percent of patients with type 2 diabetes (Young, 1993), the present study was designed to investigate the ultra structural changes occurring in the sciatic nerve in 12 weeks offspring of diabetic rats.
In common practice, the diagnosis of DPN
is based on physical examination and electrophysiology.