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DCMP is characterized by left or biventricular dilation with concomitant systolic dysfunction in the absence of extrinsic factors that may cause a similar phenotype such as coronary artery disease, arterial hypertension, valvular heart disease, or congenital heart disease.
Additionally, defective vascularization with impaired vasculogenesis and angiogenesis has also been documented in patients with DCMP  suggesting that, in addition to myocardial inflammation, microvascular ischemia may represent one of the key mechanisms involved in the development of DCMP and its progression to end-stage heart failure.
Chromosomal defects have been reported to result from consanguineous marriage and may also be associated with dCMP.[sup]
dCMP has been reported to be associated with cardioembolism, ventricular arrhythmias, and sudden cardiac death.[sup] Proband I/2 died suddenly being attributed to heart failure.[sup] Were ventricular arrhythmias, asystole, and cardioembolism excluded as alternative causes of sudden death?
For individual patient samples, we calculated bias as the difference from the DCMP creatinine concentration.
The DCMP method results (mean and expanded uncertainty) were 0.751 (0.029) mg/dL [66.4 (2.6) [micro]mol/L] and 3.850 (0.081) mg/dL [340.4 (7.2) [micro]mol/L] for the 2 concentrations, and were within the SRM uncertainty intervals.
The samples containing only DCMP and DBTDL (PPa and PPb) are compared with the unmodified PP in Fig.
The decrease in the torque in the case of the samples containing DCMP, DBTDL and different quantities of VS is faster than for the sample containing only the initiator and the catalyst, but is not so apparent.
The coaction of the A8701G mutation and genetic factors caused by consanguineous marriage might affect this family with maternally inherited hypertension and dCMP.
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