DRD2

(redirected from D2DR)

DRD2

A gene on chromosome 11q23 that encodes G protein-coupled dopamine receptor D2, which inhibits adenylyl cyclase.

Molecular pathology
Defects in DRD2 are associated with myoclonic dystonia.
References in periodicals archive ?
Predesigned primers for human PPARGC-1 (Hs01016719_m1), D2DR (dopamine 2 receptor, Hs00241436_m1), VEGF (vascular endothelial growth factor, Hs00900055_m1), MyH1 (myosin heavy chain 1, Hs00428600_m1), MyH2 (myosin heavy chain 2, Hs00430042_m1), Desmin (Hs00157258_m1), [alpha]-actinin (Hs00998100_m1), COXIV (cytochrome c oxidase subunit 4,Hs00971639_m1), vWf (Mm00550375_m1), TNF-[alpha] (tumor necrosis factor alpha, Mm00443258_m1), and ACHE (acetyl choline esterase, Hs00241307_m1) were purchased from Life Technologies.
In this scheme, we control and optimize the D2DR transmit power according to the user requirements in order to mitigate the interference.
Each D2DR accesses a random subset of the shared sub channels that are independent of the other D2DRs whereby each sub channel is accessed with equal probability [23].
We assume that the signal power received, [P.sub.S] at mUE and D2DR from the mBS and D2DS can be expressed as
The D2DS in mB[S.sub.1] communicates with D2DR at a power of [P.sub.D] using the same channel as mUE z.
where [MATHEMATICAL EXPRESSION NOT REPRODUCIBLE IN ASCII] is distributed as LN(0, [[sigma].sup.D,z).The aggregate D2DS interference for D2DR is given as
Algorithm1: PC.OVER Resource Allocation Procedure 1: Start active new mUE 2: Set t = total number of RBs, [P.sub.D2D] = [P.sup.max.sub.D2D] 3: Initialize i to 1 4: mBS checks RB i 5: If i < t then go to the next step 6: mBS checks D2DR using RB and go to step 8 7: If i > t then go to step 11 Else go back to step 4 above 8: If more than D2DRs compete for the same RB with mUE then go to next step Else check i = i+1 and go to back to step 6 9: Assign [P.sub.D2D] = [P.sup.min.sub.D2D] and go to next step 10: RB Allocation 11: End The FFR.OVER and FFR.APC schemes combine the FFR techniques and the power control mechanism.
The outage probability is defined as the probability that the SIR of the mUE or D2DR falls below the respective thresholds.
where [[gamma].sub.z,thre] and [[gamma].sub.D,thre] are the mUE and D2DR thresholds, respectively.
To address this problem, researchers have investigated methods to reduce the interference between D2D receivers (D2DRs) and mUEs in cellular networks supporting D2D communication.
Furthermore, in FFR.APC, the overall system throughput increases by limiting the cross-tier interference at the mBS below a predefined threshold level, i.e., the maximum cross-tier interference that the mBS can tolerate, by ensuring the optimization of the D2DRs power allocation for each power adjustment phase.
Because the reward pathway we previously discussed (Cunningham et al., 2012) is involved in addiction to many drugs, the dopamine-receptor encoding gene, D2DR, has been strongly implicated in a complex trait referred to as "reward deficiency syndrome" (Blum et al., 1996).