Chlorpyrifos

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A highly toxic crystalline acetylcholinesterase-inhibiting organophosphate insecticide used to control insect pests. It is a neurotoxin and suspected endocrine disruptor, and has been associated with asthma, reproductive and developmental toxicity, and acute toxicity
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This trend in dc V implied a recovering trajectory as invoked by, for example, DMT, sodium L-ascorbate, octanoic acid, chlorpyrifos-methyl, fludioxonil, and tetramethrin.
From top to bottom, each row of heat maps shows the perturbations produced by increasing concentrations of (A) octanoic acid, (B) dimethyl terephthalate, (C) chlorpyrifos-methyl, (D) butachlor, (E) dicofol, and (F) oxadiazon.
Resistance to chlorpyrifos-methyl, pirimiphos- methyl, of Rhyzopertha dominica (Coleoptera: Bostrichidae).
Two qualitatively different B-esterases from two organophosphate-resistant strains of Oryzaephilus surinamensis (Coleoptera: Silvanidae) and their roles in fenitrothion and chlorpyrifos-methyl resistance, Pestic.
granarius and Oryzaephilus surinamensis) collected during 1986 from 63 farms in South Wales, Australia were tested for resistance to malathion, fenitrothion, carbaryl, bioresmethrin, pirimiphos-methyl, chlorpyrifos-methyl and phosphine using the filter paper method (Herron, 1990).
Esterase activities toward and - naphthylacetate, p-nitrophenylacetate, methylthiobutyrate, and phenylacetate were correlated with resistance to chlorpyrifos-methyl, and pirimiphos-methyl.
general population is exposed to chlorpyrifos, chlorpyrifos-methyl, their oxons, TCPy, or a combination of these chemicals.
Oxidative desulfuration of chlorpyrifos, chlorpyrifos-methyl, and leptophos by rat brain and liver.
Monitoring for resistance to chlorpyrifos-methyl, pirimiphos-methyl and Malathion in Kansas populations of stored-product insects.
In addition, these data can help distinguish between the individual pesticides in OP pairs (e.g., methyl parathion from parathion, chlorpyrifos from chlorpyrifos-methyl).
We have presented individual and population-level urinary metabolite data indicating widespread exposure of children to the parent compounds of carbaryl or naphthalene; chlorpyrifos, chlorpyrifos-methyl, or triclopyr; and malathion.