SCIWORA can have a wide spectrum of neurological dysfunction, ranging from mild, transient spinal cord concussive deficits to permanent, complete injuries of the spinal cord6
. Approximately one-quarter of the affected children may experience delayed onset of neurologic signs ranging from 30 minutes to four days1.
The pathological lesions in JE are foci of neuronal degeneration with parenchymal and perivascular inflammatory responses are found principally in thalamus and brain stem, as well as in the hippocampus, temporal cortex, cerebellum, and spinal cord6
. These changes based upon the anatomical locations describe the most of clinical manifestations of JE.