CD96

CD96

a type I transmembrane protein present on T cells, natural killer cells, and activated B cells; primarily expressed on cellular activation, suggesting ligand-binding activity.
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(30) CD112 and CD155 are ligands for the activating receptor CD226 (DNAM-1) present on natural killer cells, (31) but also interact with inhibitory checkpoint molecules CD96 and T-cell immunoreceptor with Ig and ITim domains (TIGIT).
serine peptidase Peptide hormone processing TLL1 Tolloid like 1 Cell differentiation Troglitazone SCN4A Sodium voltage-gated Regulation of ion channel alpha subunit 4 transmembrane transport CD96 CD96 molecule Cell adhesion PCK1 Phosphoenolpyruvate Glucose metabolic process Carboxykinase 1 ADAMTS18 ADAM metallopeptidase Proteolysis with thrombospondin type 1 motif 18 MOGAT1 Monoacylglycerol Diacylglycerol O-acyltransferase 1 biosynthetic process ADRA1A Adrenoceptor alpha 1A G-protein-coupled receptor signaling pathway SCGN Secretagogin.
In the NCL blue group, BUB1B,MSR1, FANCD2, and ERCC5 were significantly upregulated, while DKC1, HFE, MSR1, FLCN, MSH6, CD96, RAD51B, ERCC5, and TP53 genes were significantly downregulated.
In the DMCL green group, only EPHB2, SDHC, and NF1 genes were significantly expressed at higher levels, while 12 genes, CD96, PTCH2, AXIN2, NF1, EPCAM, MSH6, GALNT12, ERCC5, KIT, NF1, DKC1, and TP53, had significantly lower expression.
In contrast, 9 genes were detected with significantly lower expression, DKC, HFE, MSR, FLCN, MSH6, CD96, RAD51B, ERCC5, and TP53.
On the other hand, the other 12 genes, CD96, PTCH2, AXIN2, NF1, EPCAM, MSH6, GALNT12, ERCC5, KIT, NF1, DKC1, and TP53, had lower expression (Table 3).
The team has discovered a protein known as CD96 that is found on disease-fighting white blood cells.
He discovered cancer cells express a molecule, recognised by CD96, which blocks the killer cells from reacting.
Adicionalmente, estas celulas expresan otras moleculas que funcionan como correceptores y que regulan la actividad citolitica y la produccion de citoquinas, como las moleculas NKR-P1A (CD161), CD96 y 2B4 (CD244).
Sprinkler irrigation was used to enhance grain mold in one experiment (CW96), while the other was exposed to natural conditions (CD96).
The maternal inheritance pattern of extranuclear genes contributing to grain mold resistance was estimated from the comparison of reciprocal [F.sub.1] progeny Sureno x RTx430 vs RTx430 x Sureno at CW96 and CD96. The formula to compare the means using a large number of samples was (Sincich, 1992)
Environment Sureno RTx430 1995 College Station - Sprinkle Irrigation (CW95) 2.59(a)([dagger]) 4.44(b) 1995 Beeville (BE95) 2.76(a) 4.29(b) 1996 College Station - Sprinkle Irrigation (CW96) 3.85(a) 4.91(b) 1996 College Station (CD96) 3.92(a) 4.96(b) 1997 College Station - Sprinkle Irrigation (CW97) 2.29(a) 4.51(b) 1997 College Station (CD97) 2.17(a) 4.41(b) 1997 Beeville (BE97) 1.68(a) 4.71(b) 1997 Corpus Christi (CC97) 1.98(a) 4.71(b) Combined Across Environments 2.66(a) 4.62(b) ([dagger]) Letters a, b indicate that the mean of Sureno and RTx430 are significantly different at P < 0.01.