Mentioned in ?
References in periodicals archive ?
But researchers at Boston University say they have found a new biomarker called CCL11 that may enable the disease to be recognised while the sufferer is alive.
Department of Veterans Affairs, discovered elevated levels of a protein called CCL11 in the brains of dead football players with Chronic Traumatic Encephalopathy (CTE), but not in the brains of healthy people or people with Alzheimer's disease.
Methods: Meso scale discovery system and commercial ELISA kits were used to measure the concentrations of proinflammatory cytokines interleukin (IL)-1[sz]; tumor necrosis factor-alpha (TNF-a); IL-6; and IL-17 and CC and CXC chemokines CCL2, CCL4, CCL11, CCL13, CCL17, CCL22, and CCL26 and CXCL8, CXCL9, CXCL10, and CXCL11 in bronchoalveolar lavage fluid of asthmatics and controls.
After five months, mice in various groups were treated with prednisone and fed with gluten containing and standard diet for four weeks and applied procedure to detect minor changes in paw swelling, ANA autoantibodies, CCL11, C3c, glucose level and renal damage.
61) He also showed that inflammatory factors in the blood that increase with age, such as CCL11 caused reduction in stem cell activity.
The low-frequency ARGs that correlated with canonical variables include DEFB1 with C4, KIR with C7, HLA-A with C5, CCL11 with C7, LY6D with C4, APOBEC3B with C5, and CXCR6 with C6.
genes de quimiocinas CCL5/RANTES, CCL11, CCL24, CCL26, filagrina e outros) e (d) associados a funcao pulmonar e remodelamento bronquico (ADAM-33, DPP-10 e HLA-G, dentre outros).
4] also induced expression of CCL11, CCL5, CCL3, and CXCL2, and BB exposure resulted in upregulation of CCL4.
For example, CCL11, a normal eosinophil associated chemokine, increases in plasma with age and when administered to young mice reduces neurogenesis.
Although we did not detect any SNP associated with either oxidative DNA damage or IUGR, in univariate analysis we found four genes (MBL2, CCL7, CCL11, and CCL18) modifying the incidence of LBW.
Rando's laboratory used shared circulation in mice to discover the chemical CCL11 in the blood of old mice responsible for inhibiting stem cells in the brain.
Decreased protein concentration without gene down-regulation was observed for CCL, CCL11, IL-1[alpha], and vascular endothelial growth factor (VEGF).