Nikolai, Russian pathologist, 1885-1964. See: Anitschkow cell, Anitschkow myocyte.
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References in periodicals archive ?
Anitschkow, "On experimental cholesterin steatosis and its significance in the origin of some pathological processes," Zentralblatt der Allegemeine Pathologie und Anatomie, vol.
(6) Soon that irrepressible polymath, Rudolf Virchow, reasoned that lipid buildup in artery walls caused atherosclerosis, a surmise confirmed 100 years ago by Russian pathologist Nikolai Anitschkow, who fed rabbits a lipid-dripping diet and gave them all hard arteries.
(27) Acute rheumatic heart disease can result in the formation of granulomatous lesions known as Aschoff bodies, which tend to be perivascular in location and may contain enlarged macrophages with nuclear chromatin condensed into a wavy ribbon resembling a caterpillar, that is, Anitschkow cells.
Larger mononuclear cells called Anitschkow cells may be seen within the nodule and, on cut section, bear an owl-eye appearance.
Nikolaj Nikolajewitch Anitschkow (1885-1964) established the cholesterol-fed rabbit as a model for atherosclerosis research.
1.11 ARATURUK (China) 11.1 KURUMAYA (a rickshaw puller) 1.14 ADMONITION 11.4 KENNEL-SHAPED 1.17 AKULERQOQ (Greenland) 11.7 KAWNGSUNG (Burma) 1.21 ANTROLIKU (Cyprus) 12.1 LEPIDOPTERA 1.24 ARCHICORTEX (Sted ) 12.4 LOW-CRESTED (Web2 ) 1.12 ARGUMENTAL 11.2 KRANZELERB (Austria)) 1.15 ANTIPASTO (Hors d'oeuvre) 11.5 KALEIDOSCOPE 1.18 ANTINUCLEAR 11.8 KNURRISH (gnarled) 1.22 ALEKSANDROV (Belarus) 12.2 1.25 ARTIFICIALLY 12.5 LAUNDERETTE 1.13 ALTRUISM 11.3 KURJAKOVAC (Bosnia-H.) 1.16 ALTENDONOP (Germany) 11.6 KULENKAMPFF (a violinist) 1.19 ACCUMULATES 11.9 KULLUMBRI (Albania) 1.23 ANITSCHKOW (a type of cell) 12.3 LINGUISTIC 1.26 AUNJETITZ (Czechoslovakia) 12.6 LIGHT-PROOF (Cham ) ***
Nikolajewitsch Anitschkow established the cholesterol-fed Newzealand white rabbit (NZW) as a model for atherosclerosis research on account of being most susceptible and sensitive to present atheromatous lesions, quite similar to and with the involvement of same molecular mechanisms as in humans, through high cholesterol diet7.
The current era of atherosclerosis research began in 1913 when Nikolai Anitschkow demonstrated that, when fed large amounts of cholesterol, rabbits developed vascular lesions similar to atherosclerosis seen in humans.
It is manifested by Aschoff bodies, which are discrete, commonly perivascular, areas of fibrinoid degeneration surrounded by lymphocytes, occasional plasma cells, and plump macrophages with their nuclear chromatin clumped in a wavy ribbon resembling a caterpillar (Anitschkow cells).