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The diagnosis of ARVD at its early stages remains a clinical challenge.
The structural abnormalities occurring in ARVD include RV dilatation (localised or global bulging), right ventricular outflow tract (RVOT) enlargement, thinning of the RV wall (segmental or global) and aneurysm formation.
Fat visualisation on MRI has not been found to be specific for ARVD, however, (9, 10) and there is poor inter-reader agreement on reporting of its presence and severity.
Recently, the first observations of delayed enhancement of fibrotic tissue in patients with ARVD have been reported on.
ARVD, known as one of the common causes of SCD during a sports activity, has not been found in our study.
ARVD is an inherited myocardial disease that predominantly affects the right ventricle and, less commonly, the left ventricle [8,25].
In patients with ARVD, the risk of SCD is 5.4 times higher during competitive sports than during sedentary activity [25].
In each adolescent or young individual with palpitations, syncope, aborted sudden death or ventricular tachycardia (VT) of left bundle branch block morphology, ARVD should be suspected [8,49].
We computed these indices also in small series of patients with different cardiac diseases (ARVD, left ventricular hypertrophy due to aortic stenosis, myocardial infarction with and without arrhythmias) and in some groups significant differences from normal subjects were observed.