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060 XH, Xanthine and hypoxanthine; CP, crude protein; TDN, total digestible nutrients; SEM, standard error of mean.
Therapeutic effects of xanthine oxidase inhibitors: Renaissance half a century after the discovery of allopurinol.
Xanthine oxidase inhibitors should not be discontinued in the event of an acute attack in the patient who is on treatment.
64 Values are given as the median (minimum-maximum) (Mann-Whitney U test) p: Plasma, e: Erythrocyte, MDA: Malondialdehyde, XO: Xanthine oxidase, SOD: Superoxide dismutase, F/M: Female/Male
13 CAT, catalase; GSH-Px, glutathione peroxidase; MDA, malondialdehyde; NO, nitric oxide; SOD, superoxide dismutase; XO, xanthine oxidase Values are mean [+ or -] SD (n=7) * P<0.
The xanthine group has a number of general physiological effects in humans, including central nervous system stimulation, Cardiac muscle stimulation, diuresis, bronchial, uterine, and vascular smooth muscle relaxation, peripheral and coronary vasodilation, and cerebral vasoconstriction.
ROS also are produced by a variety of oxidative enzymes present in cells, such as the previously mentioned xanthine oxidase.
The key claims in the new patent cover use of the entire family of drugs known as xanthine oxidase inhibitors applied to contractile disorders of the heart, including congestive heart failure.
Persistence of bovine milk xanthine oxidase activity after gastric digestion in vivo and in vitro.
Results: Exercise-induced increases in plasma markers of purine catabolism (hypoxanthine, xanthine oxidase and serum uric acid) and circulating cytosolic proteins (myoglobin, fatty acid-binding protein, and creatine kinase) were significantly attenuated by LCLT.