small vessel vasculitis

small vessel vasculitis

Internal medicine Vasculitis affecting vessels smaller than arteries–eg, arterioles, venules, and capillaries Clinical Palpable purpura, nodules, ulceration, urticaria; 30-50% involve GI tract and are accompanied by fever, neuritis, glomerulonephritis Examples Churg-Strauss syndrome, cutaneous leukocytoclastic angiitis, essential cryoglobulinemic vasculitis, Henoch-Schönlein purpura, microscopic polyangiitis, Wegener's granulomatosis, etc. See Systemic vasculitis.
References in periodicals archive ?
MPA is a small vessel vasculitis according to the Chapel-Hill Consensus Conference on the nomenclature of systemic vasculitis establishing the names and definitions of many vasculities that affect the kidneys (1).
Wegener's granulamotosis (WG) is a rare disease characterized by a triad of necrotizing granulomas in the upper and lower respiratory tracts, small vessel vasculitis, and glomerulonephritis.
14,15) An increase in CEC number has been recently detected in different diseases, most of which are characterized by prominent vascular pathology, such as acute coronary syndromes, (16) sickle cell anemia, antineutrophilic cytoplasmic antibody-associated small vessel vasculitis, systemic sclerosis, and SLE.
On further review they were diagnosed with polyarteritis nodosa like vasculitis and small vessel vasculitis respectively with good therapeutic response on appropriate treatment.
Thirteen percent of patients had subcutaneous nodule, which is thought to occur as a result of small vessel vasculitis with fibrinoid necrosis.
Circulating endothelial cells as markers for ANCA-associated small vessel vasculitis.
Henoch-Schonlein purpura (HSP) is a small vessel vasculitis that affects predominantly the skin, joints, gastrointestinal tract, and the kidneys (1,2).
In addition to being a powerful diagnostic tool, recent evidence suggests that ANCA may play a role in the pathogenesis of Wegener granulomatosis and other forms of small vessel vasculitis.
The focus of this article will be the treatment of pauciimmune small vessel vasculitis associated with the presence of ANCA.
The consistent demonstration of ANCA in patients with these forms of small vessel vasculitis raises the issue of their pathogenicity.
Such evidence includes the absence of disease in fetuses or newborns of pregnant women with circulating ANCA and clinical ANCA-associated small vessel vasculitis (AASV).
Studies have been designed to look at the best management of patients with more severe forms of systemic small vessel vasculitis.

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