small vessel vasculitis

small vessel vasculitis

Internal medicine Vasculitis affecting vessels smaller than arteries–eg, arterioles, venules, and capillaries Clinical Palpable purpura, nodules, ulceration, urticaria; 30-50% involve GI tract and are accompanied by fever, neuritis, glomerulonephritis Examples Churg-Strauss syndrome, cutaneous leukocytoclastic angiitis, essential cryoglobulinemic vasculitis, Henoch-Schönlein purpura, microscopic polyangiitis, Wegener's granulomatosis, etc. See Systemic vasculitis.
References in periodicals archive ?
Henoch-Schonlein purpura (HSP) is the most common form of systemic small vessel vasculitis involving the skin, joints, kidneys, and gastrointestinal tract in childhood.
Introduction: IgA vasculitis is a systemic small vessel vasculitis accompanied by gastrointestinal tract (GIT) and renal involvement.
One of the main areas of revision included the description of 2 pathophysiologically different groups of small vessel vasculitis including immune complex vasculitis and AAV (Table 1).
The histological manifestations include parenchymatous necrosis, small vessel vasculitis and granulomatous inflammation not vernix (2).
Symmetric bilateral basal ganglia lesions on neuroimaging can be probably caused by carbon monoxide intoxication, hypoxia, toxins, metabolic disorders, small vessel vasculitis, or infection, but these lesions usually do not regress spontaneously [4].
Infective endocarditis by Bartonella quintana masquerading as antineutrophil cytoplasmic antibody-associated small vessel vasculitis," Cardiology, vol.
Henoch Schonlein purpura, also known as Immunoglobulin A vasculitis (IgAV) is a small vessel vasculitis associated with IgA deposition in the affected organs.
Granulomatosis with polyangiitis (GPA) previously known as Wegner's granulomatosis, is a small vessel vasculitis that preferentially involves capillaries, arterioles and venules, presenting as multisystemic disease classically with alveolar haemorrhage and renal insufficiency.
The characteristics which differentiate antineutrophilic cytoplasmic antibody-related vasculitis from small vessel vasculitis include being necrotizing vasculitis, absence of immune complexes (or presence of very few immune complexes) and being related with PR3 and MPO-ANCA.
RESULTS: 20 patients who were clinically diagnosed as cutaneous small vessel vasculitis and histologically confirmed as leucocytoclastic vasculitis were included in the study and all parameters were evaluated.
Cutaneous leukocytoclastic angiitisis (hypersensitivity vasculitis) is a clinical term that generally refers to an Immune Complex-mediated small vessel vasculitis of the skin that spares internal organs and usually follows drug exposures or infections.
The researchers divided the patients into five subtypes: cutaneous small vessel vasculitis (CSW), immunoglobulin A (IgA) vasculitis, urticarial vasculitis, cryoglobulinemic vasculitis, and antineutrophil cytoplasmic antibody-associated vasculitis.

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