secondary hyperaldosteronism


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secondary hyperaldosteronism

excessive production of aldosterone caused by an extraadrenal disorder, such as heart failure, kidney disease, cirrhosis, or hypoproteinemia.
References in periodicals archive ?
Renin and aldosterone are characteristically increased (but are sometimes normal), and the aldosterone/ renin ratio is normal or low, in cases of secondary hyperaldosteronism that may occur in hypertensive patients who have been treated with thiazide diuretics and patients with renovascular hypertension or the rare reninoma.
Clinical studies do not show a loss of effect to laxatives (Muller Lissner, 1993) and studies carried out in rats also suggest that long-term sennoside treatment in diarrhogenic doses does not induce habituation in the sense of a reduced laxative effect and does not lead to secondary hyperaldosteronism (Leng-Peschlow et al.

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