secondary brain injury


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Related to secondary brain injury: primary brain injury

secondary brain injury

Brain damage due to cellular disruption, electrolyte disarray, inflammation, insufficient oxygen, or vasospasm after head trauma.
See also: injury
References in periodicals archive ?
For the foreseeable future, improved patient outcomes will depend on organised trauma response systems, particularly to prevent the potentially reversible effects of secondary brain injury.
This finding is supported in the literature, as ICU nurses often rely on additional physiological parameters such as oxygen saturation, brain oxygenation, and cerebral perfusion pressure when making decisions about interventions to prevent secondary brain injury (McNett et al.
Judgments of critical care nurses about risk for secondary brain injury.
Brain injury is commonly discussed and treated as being either a primary or secondary brain injury.
Nursing judgments about appropriate interventions when managing secondary brain injury in critically ill TBI patients are influenced by worsening values for oxygen saturation, intracranial pressure (ICP), cerebral perfusion pressure (CPP), and nursing shift.
In fact, up to 35% of patients develop secondary brain injury within days after the initial trauma (Wright, 2005).
In conjunction with cellular energy depletion, these processes influence the severity of secondary brain injury (Tavazzi et al.
Secondary brain injury contributes to long-term neurological sequelae.
To reduce morbidity and mortality, the NICU team must be vigilant in its efforts to recognize and treat secondary brain injury.
In fact, as many as 90% of patients who die from TBI show some evidence of secondary brain injury (Shackford et al.
Prevention of primary and secondary brain injury is always an important nursing intervention in this population.
An important treatment consideration of neonates involves prevention of a primary or secondary brain injury.