reoxygenation

reoxygenation

 [re-ok″sĭ-jĕ-na´shun]
in radiobiology, the phenomenon in which hypoxic (and thus radioresistant) tumor cells become more exposed to oxygen (and thus more radiosensitive) by coming into closer proximity to capillaries after death and loss of other tumor cells due to previous irradiation.

reoxygenation

/re·ox·y·gen·a·tion/ (re-ok″sĭ-jen-a´shun) in radiobiology, the phenomenon in which hypoxic (and thus radioresistant) tumor cells become more exposed to oxygen (and thus more radiosensitive) by coming into closer proximity to capillaries after death and loss of other tumor cells due to previous irradiation.

reoxygenation

in radiobiology, the phenomenon in which hypoxic (and thus radioresistant) tumor cells become more exposed to oxygen (and thus more radiosensitive) by coming into closer proximity to capillaries after death and loss of other tumor cells due to previous irradiation.
References in periodicals archive ?
Roudier, Bachelet, and Perrin "Pyruvate reduces DNA damage during hypoxia and after reoxygenation in heptocellular carcinoma cells.
Upon re-entry to the pulmonary circulation and subsequent reoxygenation of HbS, sickle cells revert to their original shape.
Extracellular signal-regulated protein kinase activation during reoxygenation is required to restore ischaemia-induced endothelial barrier failure.
Influence of hypoxia and reoxygenation on cytokine-induced production of proinflammatory mediators in articular cartilage.
Furthermore, the rate and degree of subsequent mPTP opening during reoxygenation is proportionally related to the severity of the initial ischaemic insult (14).
After the incubation with DG extract at 30 or 100 [micro]g/ml for 24 h, cells were then subjected to 2 h of hypoxia and followed by 16 h of reoxygenation, as previously described (Chiu et al.
Protection of cultured myocardial cells from anoxia and reoxygenation induced injuries of cell membrane, endoplasmic reticulum, and mitochondria (Ye et al.
Reoxygenation was achieved by adding another 2 ml of incubation medium, which was air saturated, to the incubation chamber (Schild et al.
Two parameters derived from NIRS data are of potential interest in PFM training: the recovery interval of muscle oxygenation and the muscle reoxygenation rate.
They preconditioned mice by exposing them to varying low levels of oxygen for six hours followed by four hours of reoxygenation.
Intermittent hypoxia followed by complete reoxygenation selectively activates the pro-inflammatory transcription factor NF[kappa]B, and increases circulating tumour necrosis factor (TNF)-alpha in OSA patients.