renal infarction

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renal infarction

Nephrology Death of renal tissue, usually caused by renal artery sternosis


1. the formation of an infarct.
2. an infarct.

cardiac infarction
see myocardial infarction (below) and also myocardial infarction.
cerebral infarction
an ischemic condition of the brain, causing a persistent focal neurological deficit in the area affected.
infarction fever
an aseptic fever caused by liberation of pyrogens from damaged tissue.
intestinal infarction
a common occurrence in horses due to occlusion of arteries by larvae of Strongylus vulgaris. Sections of intestine, sometimes very large ones, become devitalized leading to peritonitis and death.
May also result from torsion or strangulation. See also thromboembolic colic.
myocardial infarction
gross necrosis of the myocardium, due to interruption of the blood supply to the area. See also myocardial infarction.
pulmonary infarction
localized necrosis of lung tissue, due to obstruction of the arterial blood supply.
renal infarction
is usually conical, anemic and multiple and may heal leaving a narrow scar. It is usually clinically inapparent unless the obstructing material is infected. This leads to the development of renal abscess or embolic nephritis, also usually without clinical signs unless the abscesses are large or numerous.
spinal cord infarction
caused sometimes by fibrocartilaginous emboli of prolapsed disk material, causing sudden loss of function of large sections of the spinal cord, leading to flaccid paralysis of the hindlimbs or of all four, depending on the site of the infarct.
splenic infarction
usually hemorrhagic; may be difficult to differentiate from subcapsular hematoma.
venous infarction
a thrombus in a vein may cause infarction, e.g. in the thigh muscles of downer cow, recumbent for long periods, or in the gastric mucosa of pigs, where it is a common finding in acute septicemia.
References in periodicals archive ?
Acute renal embolism: Forty-four cases of renal infarction in patients with atrial fibrillation.
Renal infarction in the ED: 10-year experience and review of the literature.
According to the literature, the clinical diagnosis of acute renal infarction is usually based on typical clinical findings that are nonspecific and increased risk for thromboembolism, (2,4,5) a profile that was present in our patient.
Serum LDH as a characteristic marker for cell necrosis is known to be elevated in patients with acute renal infarction.
Angiography, renal scintigraphy, intravenous pyelography, ultrasonography, and enhanced CT may be useful in diagnosing acute renal infarction ante mortem.
Although Domanovits et al (5) found signs of slightly impaired renal function in only 6 of their 17 patients after 7 days, we stress that early diagnosis of acute renal infarction is mandatory to establish effective acute and long-term therapy for the preservation of renal function.