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prerenal azotemia

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azotemia
an excess of nitrogen-containing compounds in the blood. See also uremia.

postrenal azotemia
is caused by reduced renal blood flow caused by increased pressure within the renal collecting system, e.g. hydronephrosis and urine retention from a variety of causes.
prerenal azotemia
is due to extrarenal causes that reduce renal blood flow and glomerular filtration, e.g. dehydration, shock, reduced cardiac output, decreased plasma albumin osmotic pressure.
primary renal azotemia
results from loss of renal functional parenchyma.

prerenal azotemia
Renal underperfusion Nephrology The most common form of acute renal failure, characterized by ↑ nitrogenous waste, due to ↓ blood flow to the kidney Lab ↑ nitrogenous wastes–eg, creatinine and urea, which act as poisons when they accumulate in the body, damaging tissues and compromising organ function Risk factors ↓ blood volume–eg, dehydration, prolonged vomiting, diarrhea, bleeding, burns, etc; pump failure–eg, CHF, shock, kidney trauma or surgery, renal artery embolism, and other types of renal artery occlusion. See Renal failure.


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These values include a significant drop in the hemoglobin level attributable to active bleeding, prerenal azotemia due to renal hypoperfusion during the early stages of decreased cardiac output (Russo et al.
19) Although diuretics are effective treatment, they may exacerbate prerenal azotemia.
Renal injury that appears after acute high-dose lead exposure may include reversible deficits in proximal tubular reabsorption and prerenal azotemia induced by renal vasoconstriction and/or volume depletion (Coyle et al.
 
 
 
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