polymorphonuclear


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Related to polymorphonuclear: Polymorphonuclear leukocytes, Polymorphonuclear cells

polymorphonuclear

 [pol″e-mor″fo-noo´kle-ar]
having a nucleus so deeply lobed or so divided as to appear to be multiple; see under leukocyte.

pol·y·mor·pho·nu·cle·ar

(pol'ē-mōr'fō-nū'klē-ăr),
Having nuclei of varied forms; denoting a variety of leukocyte.
[G. polymorphos, multiform, + L. nucleus, kernel]

polymorphonuclear

/poly·mor·pho·nu·cle·ar/ (-noo´kle-er) having a nucleus so deeply lobed or so divided as to appear to be multiple.

polymorphonuclear

(pŏl′ē-môr′fə-no͞o′klē-ər, -nyo͞o′-)
adj.
Having a lobed nucleus. Used especially of neutrophil white blood cells.
n.
A polymorphonuclear cell.

polymorphonuclear

[pol′ēmôr′fōno̅o̅′klē·ər]
Etymology: Gk, polys + morphe + L, nucleus, nut kernel
having a nucleus with a number of segments connected by fine threads of nuclear membrane.

pol·y·mor·pho·nu·cle·ar

(pol'ē-mōr'fō-nū'klē-ăr)
Having nuclei of varied forms; denoting a variety of leukocyte.
[G. polymorphos, multiform, + L. nucleus, kernel]

polymorphonuclear

Having a lobed nucleus of varying lobe number or shape. The term is used of three classes of LEUKOCYTES, neutrophil, basophil and eosinophil.

polymorphonuclear

1. having a nucleus so deeply lobed or so divided as to appear to be multiple.
2. a polymorphonuclear leukocyte.

polymorphonuclear neutrophil (PMN)
References in periodicals archive ?
Inhibition of eicosanoid formation in human polymorphonuclear leukocytes by high concentrations of magnesium ions.
Assembly of staphylococcal leukocidin into a pore-forming oligomer on detergent-resistant membrane microdomains, lipid rafts, in human polymorphonuclear leukocytes.
Assay method for myeloperoxidase in human polymorphonuclear leukocytes.
A punch biopsy from lesions on the forearms revealed prominent edema on the upper dermis and intense cellular infiltration, and predominantly lymphocytes, polymorphonuclear neutrophils and leucocytoclasis on the perivascular areas.
Magnesium's functions include effects on platelet aggregation, vasospasm, and release of inflammatory mediators, while feverfew inhibits prostaglandin synthetase, 5-lipoxygenase, and cyclooxygenase in leukocytes and serotonin secretion in platelets and polymorphonuclear leukocytes.
Additionally, the FDP40, SDP40 (prepared from 40% v/v ethanol added of CSD) and SDP80 reduced the total leukocyte and polymorphonuclear cell migration in the pleural cavity.
Attacks of FIVIF are associated with various markers of inflammation, which are reflected: a) clinically by fever and pain in the affected sites b) histologically by an invasion of polymorphonuclear leukocytes to the serosal membranes c) serologically by activation of the cytokine cascade with elevated levels of IL-6 and soluble receptors of TNF and particularly by the increased production of the acute phase plasma proteins, fibrinogen, CRP, serum Amyloid A and phospholipase A2 (2).
Prothrombin activity and serum concentrations of albumin, total bilirubin, and alanine aminotransferase were measured, and ascitic fluid polymorphonuclear (PMN) cells were counted.
The currently accepted histologic definition of silent PID (five or fewer polymorphonuclear leukocytes and one or fewer plasma cells per 40X) was not indicative of C.
Similarly, there were no significant effects of treatment on the volume of nasal secretions, on polymorphonuclear leukocyte or interleukin-8 concentrations in nasallavage specimens, or on quantitative-virus titer.
Pathologically, an acute inflammatory reaction is seen in the periosteum along with infiltration of the connective tissues by polymorphonuclear leukocytes.
1,2) There is a prominent inflammatory element with infiltrates of polymorphonuclear leukocytes, and oftentimes a band of lymphocytes is noted peripheral to the advancing tumor margin.

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