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pneumoconiosis
(redirected from pneumoconiotic)

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pneumoconiosis /pneu·mo·co·ni·o·sis/ (-ko″ne-o´sis) deposition of large amounts of dust or other particulate matter in the lungs, causing a tissue reaction, usually in workers in certain occupations and in residents of areas with excessive particulates in the air; there are many types, including anthracosis, asbestosis, bituminosis, and silicosis.
coal workers' pneumoconiosis  black lung; a form caused by deposition of coal dust in the lungs, usually characterized by centrilobular emphysema. Different varieties of coal have different risks; bituminosis is usually more severe than anthracosis.
talc pneumoconiosis  talcosis; a type of silicatosis caused by the inhalation of talc; prolonged exposure may result in pulmonary fibrosis.

pneu·mo·co·ni·o·sis (nm-kn-ss)
n. pl. pneu·mo·co·ni·o·ses (-sz)
A disease of the lungs, such as asbestosis or silicosis, caused by long-term inhalation of particulate matter, especially mineral or metallic dust.

Pneumoconiosis (plural, pneumoconioses)
Any chronic lung disease caused by inhaling particles of silica or similar substances that lead to loss of lung function.
Mentioned in: Silicosis

pneumoconiosis
[no̅o̅′mōkō′nē·ō′sis]
Etymology: Gk, pneumon + konis, dust, osis, condition
any disease of the lung caused by chronic inhalation of dust, usually mineral dust of occupational or environmental origin. Some kinds of pneumoconioses are anthracosis, asbestosis, silicosis.

pneumoconiosis [noo″mo-ko″ne-o´sis]
any of a group of lung diseases resulting from inhalation of particles of industrial substances, particularly inorganic dusts such as the dust of iron ore or coal, and permanent deposition of substantial amounts of such particles in the lungs. The diseases vary in severity but all are occupational diseases, acquired by workers in the course of their jobs. Symptoms include shortness of breath, chronic cough, and expectoration of mucus containing the offending particles.

Silicosis is probably the best known and most severe of these diseases. asbestosis, caused by inhalation of asbestos fibers, is probably second only to silicosis in severity. Prevention and early diagnosis are important, for no effective treatment is available. Coal workers' pneumoconiosis, or black lung, usually in the form of bituminosis or anthracosilicosis, is caused by the inhalation of coal dust, often with silica, and is similar in its development and its effects to silicosis. Berylliosis is a variety found in workers exposed to beryllium in the manufacture of fluorescent lamps, and in members of their families who are contaminated by the chemicals in the worker's clothing. Other types of pneumoconiosis include aluminosis, cadmiosis, and siderosis.
talc pneumoconiosis a type of silicatosis caused by the inhalation of talc; symptoms include shortness of breath, cough, fatigue, weakness, and weight loss. Prolonged exposure may result in pulmonary fibrosis. Called also talcosis.

pneumoconiosis (noo´mōkō´nēō´sis),
any disease of the lung caused by chronic inhalation of dust, usually mineral dusts of occupational or environmental origin. The principal agents include coal, cotton, sand, and asbestos.
Pneumocystis jiroveci
n an opportunistic infection found in immunocompromised patients such as those with AIDS.

pneumoconiosis
any of a group of lung diseases resulting from inhalation of particles of industrial substances, such as the dust of iron ore or coal, and permanent deposition of substantial amounts of such particles in the lungs.
The disease is rare in animals unless there is an unusual exposure to a dusty environment. Horses are the ones most commonly exposed, usually in a mining operation (pit ponies). Exercise intolerance and chronic cough are the cardinal signs.


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interpreted their findings of high black pigment scores in liver and spleen of coal miners differently--namely, as migration of coal dust in the pneumoconiotic lung into pulmonary lymphatics and then to the systemic circulation, and also as migration of coal mine dust-laden macrophages through the walls of pulmonary blood vessels.
Using the linear fit of CWP prevalence and the calculated BAI in the seven coal mine regions, we have derived and mapped the pneumoconiotic potencies of 7,000 coal samples.
We applied standard diagnostic criteria for the recognition of pneumoconiotic lesions (7-10).
 
 
 
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