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pheresis

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pheresis /phe·re·sis/ (fĕ-re´sis) apheresis.
phe·re·sis (f-rss, fr-)
n.
Apheresis.

pheresis.
See apheresis.

apheresis [af″ĕ-re´sis]
any procedure in which blood is withdrawn from a donor, a portion (such as plasma, leukocytes, or platelets) is separated and retained, and the remainder is retransfused into the donor. Types include erythrocytapheresis, leukapheresis, lymphocytapheresis, plasmapheresis, and plateletpheresis.. Called also hemapheresis and pheresis.
therapeutic apheresis separation of whole blood into its major components and removal of the abnormal, pathogenic component. Types include plasma exchange (plasmapheresis), removal of white blood cells (leukapheresis), removal of platelets (thrombocytapheresis), and removal of red blood cells erythrocytapheresis). The process is currently used as measure of last resort when conventional therapies are unsuccessful in controlling a chronic, debilitating, or potentially fatal disease. Its primary purpose is to modify the pathologic process so that other treatments can be more effective. It is not a cure. Plasmapheresis may be used in treatment of rheumatoid arthritis, myasthenia gravis, systemic lupus erythematosus, and some malignancies, in which plasma constituents can interfere with the function of the immune system. Other diseases for which therapeutic apheresis might be used include certain blood dyscrasias such as thrombocytosis, polycythemia vera, and sickle cell anemia.

pheresis
any procedure in which blood is withdrawn from a donor, a portion (plasma, leukocytes, etc.) is separated and retained, and the remainder is retransfused into the donor. It includes plasmapheresis, leukapheresis, etc.

pheresis


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Today, MIMA's monthly transfusion average is 150 leukoreduced RBCs, 15 platelet pheresis units, and two to four fresh-frozen plasmas.
Apheresis (or pheresis or hemapheresis) is a procedure in which blood is drawn from a donor and separated into components, some of which are retained, such as plasma, and the remainder is returned by transfusion to the donor.
A variety of different agents and techniques such as methylene blue (nitric oxide inhibitor), sympathicomimetic agents, somatostatin analogues, exchange plasma pheresis and physical occlusion of intrapulmonary vascular dilatations have been employed in attempts to treat HPS, although none should be recommended for use at present.
 
 
 
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