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Conclusion: According to characteristic hearing dysfunction, AN may occur in the afferent pathway of acoustic nerve, probably accompanied by the pathological changes of efferent nerve in the olivocochlear system inside the brainstem.
Separating the contributions of olivocochlear and middle ear muscle reflexes in modulation of distortion product otoacoustic emission levels.
Effects of Type 2 Diabetes on Otoacoustic Emissions and the Medial Olivocochlear Reflex.
11,32) The difference has been attributed to the fact that the outer hair cell properties are considered to be mature at 32 weeks of gestation, (33) while it has been proposed that maturation of the medial olivocochlear efferent innervation of the outer hair cells occurs at 37 weeks.
The auditory olivocochlear (OC) efferent nerve pathway begins in the brainstem and projects to the inner ear.
Evaluation of outer hair cell function and medial olivocochlear efferent system in patients with type II diabetes mellitus.
Olivocochlear bundle stimulation affects cochlear biomechanical nonlinearity.
The medial olivocochlear tract modulates the movement of the outer hair cells by the release of acetylcholine in the synaptic cleft, causing a hyperpolarization, which is opposed to the depolarization induced by sound stimuli.
The olivocochlear efferent pathway has been studied extensively in attempts to characterize its role in hearing.
Besides, another study observed that people with tinnitus may present alteration of medial olivocochlear tract function, observed through lower otoacoustic emissions suppression [14].