Pertaining to the sympathetic pathway to the eye, damage to which produces Horner syndrome.


Pertaining to the sympathetic pathway to the eye, damage to which produces Horner syndrome
References in periodicals archive ?
Raeder syndrome (paratrigeminal oculosympathetic syndrome) is a rare clinical entity characterized by ipsilateral trigeminal sensory deficits, ptosis, and miosis, with an absence of anhidrosis secondary to interruption of the postganglionic oculosympathetic pathway.
Traumatic pseudoaneurysm of the internal carotid artery presenting with oculosympathetic palsy.
Oculosympathetic palsy or Horner's syndrome is the triad of meiosis, ptosis, and anhidrosis that results from disruption of the sympathetic pathways between the brain and the eye.
The syndrome of painful ophthalmoplegia consists of periorbital or hemicranial pain, combined with ipsilateral ocular motor nerve palsies, oculosympathetic paralysis, and sensory loss in the distribution of the ophthalmic and occasionally the maxillary division of the trigeminal nerve.
In 1971, hydroxyamphetamine (Paredrine) was readily available and widely used and therefore chosen as the drug of choice to distinguish between postganglionic and preganglionic or central causes of oculosympathetic palsy (OSP).
6,7) These have been ascribed to NA stores in postganglionic oculosympathetic terminals that are probably not yet depleted in the acute phase of the syndrome.
While any of the 1st, 2nd or 3rd-order oculosympathetic neurons may be involved, the co-existence of cerebral, brainstem, spinal cord, brachial plexus, carotid, cavernous sinus or orbital signs aid enormously in localisation of lesions.