occipital cortex

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Related to occipital cortex: Parietal cortex, temporal cortex

cortex, occipital 

The superficial grey matter on the posterior part of each hemisphere comprising Brodmann's areas 17, 18 and 19. See visual area.
References in periodicals archive ?
As illustrated in Figure 10, the power on the left brain hemisphere increases from 2 s when the left hand MI task began to be executed, especially from 15 Hz to 30 Hz, at channel C3, and at the same time, the high power appears at channels on the occipital cortex, especially Oz, at 7 Hz and 14 Hz.
Also in children and adolescents with BD, Cre levels were normal in the frontal cortex (81), posterior cingulate and occipital cortex (80).
Accordingly, reductions were seen in the occipital cortex and midbrain, whereas an increase was obtained in the temporal cortex (Figure 1B).
PET studies have shown activation of the superior occipital cortex, the inferior parietal cortex and the premotor cortex during the mental construction of 3D images; no activation of the primary visual areas was observed (Mellet, Tzourio, Crivello, Joliot, Denis & Mazoyer, 1996); failing to provide evidence that primary visual cortex is engaged in the generation of visual images (Cocude, Mellet, & Denis, 1999).
There is a little deactivation in the medial occipital cortex extending up into the medial parietal cortex, though not as strikingly as in the hard versus null.
Our previous work in the field of visual attention and attention deficit hyperactivity disorder indicated that visual attention is associated with increased processing speed at the occipital cortex near the primary visual cortex (Silberstein et al.
They also examined synapses in the occipital cortex, an area involved in simple visual perception.
They found that poor imagers, classified according to their performance on two spatial tasks, the Minnesota Paper Form Board and the Mental Rotation Test (MRT) (Vandenberg & Kuse, 1978), showed a global increase in rCBF during the task whereas in good imagers increases in rCBF were localized particularly in the inferior parietal and occipital cortex.
Indeed if Haksar had thought more about this he would have realised that split-brain experiments on visual perception could not work as they do unless the retina of each eye is connected to occipital cortex of both hemispheres.
The following structures were sampled: frontal lobe (superior middle and inferior frontal gyri), cingulate gyrus, temporal lobe (superior, middle and inferior gyri), parietal lobe (inferior and superior portions), thalamus, caudate, putamen, occipital cortex and cerebellum.
The visual disturbance will move with the eyeball if it originates in the retina, but will remain fixed if it originates in the occipital cortex.
In contrast, alexia without agraphia (which is associated with lesions to the left occipital lobe and the splenium of the corpus callosum) was thought to arise from a disconnection of visual word form processing ( in the left angular gyrus) from visual processing in the occipital cortex.