neoplasia

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neoplasia

 [ne″o-pla´zhah]
the formation of a neoplasm.
cervical intraepithelial neoplasia (CIN) dysplasia of the cervical epithelium, often premalignant, characterized by various degrees of hyperplasia, abnormal keratinization, and the presence of condylomata.
multiple endocrine neoplasia (MEN) a group of rare hereditary disorders of autonomous hyperfunction of more than one endocrine gland. In Type I (MEN I), called also Wermer's syndrome, there are tumors of the pituitary, parathyroid gland, and pancreatic islet cells in association with a high incidence of peptic ulcer. Type II (MEN II), called also Sipple's syndrome, is characterized by medullary carcinoma of the thyroid, pheochromocytoma, often bilateral and multiple, and parathyroid hyperplasia. Type III (MEN III), called also mucosal neuroma syndrome, resembles Type II except that parathyroid hyperplasia is rare, the mean survival time is shorter, and there may be neuromas and neurofibromas. All forms are transmitted as autosomal dominant traits.

ne·o·pla·si·a

(nē'ō-plā'zē-ă),
The pathologic process that results in the formation and growth of a neoplasm.
[neo- + G. plasis, a molding]

neoplasia

/neo·pla·sia/ (-pla´zhah) the formation of a neoplasm.
cervical intraepithelial neoplasia  (CIN) dysplasia of the cervical epithelium, often premalignant, characterized by various degrees of hyperplasia, abnormal keratinization, and the presence of condylomata.
gestational trophoblastic neoplasia  (GTN) a group of neoplastic disorders that originate in the placenta, including hydatidiform mole, chorioadenoma destruens, and choriocarcinoma.
multiple endocrine neoplasia  (MEN) a group of rare diseases caused by genetic defects that lead to hyperplasia and hyperfunction of two or more components of the endocrine system; type I is characterized by tumors of the pituitary, parathyroid glands, and pancreatic islet cells, with peptic ulcers and sometimes Zollinger-Ellison syndrome; type II is characterized by thyroid medullary carcinoma, pheochromocytoma, and parathyroid hyperplasia; type III is similar to type II but includes neuromas of the oral region, neurofibromas, ganglioneuromas of the gastrointestinal tract, and café-au-lait spots.

neoplasia

(nē′ō-plā′zhə)
n.
1. Formation of new tissue.
2. Formation of a neoplasm or neoplasms.

neoplasia

[nē′ōplā′zhə]
Etymology: Gk, neos + plassein, to mold
the new and abnormal development of cells that may be benign or malignant. neoplastic [-plas′tik] , adj.

neoplasia

 Oncology Abnormal and uncontrolled cell growth. See Anal intraepithelial neoplasia, Cervical intraepithelial neoplasia, Ductal intraepithelial neoplasia, Hereditary neoplasia, Hereditary preneoplasia, Papillary neoplasia, Prostatic intraepithelial neoplasia, Vulvar intraepithelial neoplasia.

ne·o·pla·si·a

(nē'ō-plā'zē-ă)
The pathologic process that results in the formation and growth of a neoplasm.
[neo- + G. plasis, a molding]

neoplasia

The process of tumour formation.

Neoplasia

Abnormal growth of cells, which may lead to a neoplasm, or tumor.
Mentioned in: Pap Test

neoplasia

abnormal cell proliferation generating tissue that is characterized by rapid and non-controlled cell division, poor cellular differentiation, and which is potentially cancerous

ne·o·pla·si·a

(nē'ō-plā'zē-ă)
The pathologic process that results in formation and growth of a neoplasm.
[neo- + G. plasis, a molding]

neoplasia (nē´ōplā´zhə),

n the disease process responsible for neoplasm formation. See also neoplasm.

neoplasia

the formation of a neoplasm.

Patient discussion about neoplasia

Q. What is a brain tumor?

A. A brain tumour is any intracranial tumor normally either in the brain itself in the cranial nerves, in the brain envelopes, skull, pituitary and pineal gland, or spread from cancers primarily located in other organs (metastatic tumors). It is created by abnormal and uncontrolled cell division. Primary (true) brain tumors (which start in the brain) are commonly located in the posterior cranial fossa in children and in the anterior two-thirds of the cerebral hemispheres in adults, although they can affect any part of the brain.

Q. Is this a tumor? I felt a lump in my breast a few days ago in the shower. Is this a Tumor? Help! I'm scared.

A. If you felt a lump in your breast then you should go see your Doctor to check whether or not it is something that could be dangerous.

Q. what is carcinoid tumors? I had my appendix removed and the doctor came in the room very shocked and said it was full of carcinoid tumors. Im scared to get them somewhere else.

A. ya I have pain all the time but the doctors wont give me anything cuz im so young they don't want me hooked on anything. thank you sooo much for being so kind.

More discussions about neoplasia
References in periodicals archive ?
The spectrum of neoplastic cells found in MIFS varies within and among tumors.
The predominant population of neoplastic cells were polygonal with distinct cell borders, a moderate amount of eosinophilic to amphophilic lacy cytoplasm, and an irregularly round to oval nucleus possessing vesiculate chromatin and 1-2 prominent nucleoli.
Recent studies have demonstrated intact E-Cadherin expression in the neoplastic cells, suggesting that these are actually representing a variant of ductal carcinoma with lobular growth patterns.
Our data showed that in all cases where higher number (more than 1%) of neoplastic cells exhibited class III [beta]-tubulin expression, this was detected by both TU-20 and TuJ-1.
An immersion objective was used to count NORs present in 30 nuclei of randomly chosen neoplastic cells.
Research reveals that these therapies induce cells to death by apoptosis, once both treatments damage the DNA of neoplastic cells, activating the p53 gene (FISHER, 2001).
36 (Biogenics, Karnataka, India), and vimentin (Yentana, Tucson, AZ, USA) was negative in the main population of neoplastic cells.
The flow cytometric study of the peripheral blood showed that the circulating neoplastic cells (comprising about 40% of total white blood cells) seemed to be of NK-cell lineage with expression of CD45 (bright), CD2, CD56, CD11b, CD16 (partial), and cytoplasmic CD3, but the sample was negative for surface CD3, CD57, CD4, CD5, CD7, CD8, TCR, CD123, terminal deoxynucleotidyl transferase, CD34, B-cell markers, or myeloid markers (Figure 2, A through F).
Neoplastic cells were negative for calponin, ruling out myoepithelial cells.
4,6 This treatment faces the challenge posed by the morpholocal and genetic hetergonecity that tumour cells flaunt, however using microarray technologies and proteomics, together with transcriptional targeting, its ability to preferentially target neoplastic cells and spare the normal ones.
The neoplastic cells may assume bizarre shape and large cell size.