neisserial

neisserial

 [nīs-se´re-al]
of, relating to, or caused by Neisseria.
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References in periodicals archive ?
By flow cytometry, the outbreak strain had low expression of factor H binding protein and low binding with antisera to the Neisserial heparin binding antigen included in Bexsero (Table) (7).
MenB-4C consists of three recombinant proteins (neisserial adhesion A [NadA], factor H binding protein [FHbp] fusion protein, and neisserial heparin binding antigen [NHBA] fusion protein) and outer membrane vesicles (OMVs) containing outer membrane protein PorA serosubtype P1.
Bexsero[R] contains four immunodominant antigens: fHbp, PorA, NadA and neisserial heparin binding antigen (NHBA) (Bai et al, 2011).
The septic shock caused by Neisserial invasive disease is characterized by the presence of skin lesions (petecchial rashes and purpura).
Other known genes in this pathway, however, are absent from the sequenced neisserial genomes.
Because serogroup distributions vary by country, the vaccine in the United Kingdom protects against group C Neisserial meningitidis, while the U.
Deficiency of the complement component, C3, leads to repeated sinopulmonary infections similar to agammaglobulinemia, while deficiencies in the terminal complement components (C6, C7, C8, C9) result in recurrent systemic infections or meningitis with neisserial organisms.
Bexsero includes 4 different components: an FHbp of variant 1 (subfamily B); a Neisseria adhesion A protein (NadA); a neisserial heparin-binding antigen (NhbA); and outer membrane vesicles from a serogroup B strain containing PorA P1.
MenB-4C consists of three recombinant proteins (Neisserial adhesin A [NadA], factor H binding protein [FHbp] fusion protein, and Neisserial Heparin Binding Antigen [NHBA] fusion protein), and outer membrane vesicles (OMVs) containing outer membrane protein PorA serosubtype P1.
Multilocus sequence typing and genotypic analysis of PorA variable regions (VRs) (porA VR1 and VR2), ferric enterochelin receptor VR (fetA), neisserial heparin-binding antigen (nhba), neisserial adhesin A (nadA), and factor H binding protein (fhbp) encoding regions were performed by using described protocols (11,12).
The most well-known example is the susceptibility of patients with terminal complement component deficiencies to neisserial infections.